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Titlebook: Helicobacter pylori; Hidekazu Suzuki,Robin Warren,Barry Marshall Book 2016 Springer Japan 2016 H. pylori infection.MALT lymphoma.eradicati

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發(fā)表于 2025-3-21 19:49:06 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Helicobacter pylori
編輯Hidekazu Suzuki,Robin Warren,Barry Marshall
視頻videohttp://file.papertrans.cn/426/425493/425493.mp4
概述Reviews the most recent pathogenetic, diagnostic, and therapeutic approaches in connection with Helicobacter pylori.Maximizes reader insight into the trends in clinical practice to address Helicobacte
圖書封面Titlebook: Helicobacter pylori;  Hidekazu Suzuki,Robin Warren,Barry Marshall Book 2016 Springer Japan 2016 H. pylori infection.MALT lymphoma.eradicati
描述This book provides a detailed review of the most recent pathogenetic, diagnostic and therapeutic approaches in connection with Helicobacter pylori, along with priority areas in research and trends in clinical practice to address this significant pathogen acknowledged as the causative agent in a wide range of diseases, including gastric adenocarcinoma, MALT lymphoma and immune thrombocytopenic purpura (ITP). Medical authorities worldwide now recommend a “test and treat” strategy for this bacterial infection. Eradication therapy approaches range from general mass eradication therapy to tailor-made therapy for antibiotic-resistant refractory infection. In regions with high prevalence of H. pylori infection, the infection itself would be considered a major parameter to predict the risk of gastric adenocarcinoma development. The main interest has shifted recently to the risk stratification strategy for gastric cancer development and to the wide-ranging application of its serological prevention program..This book will benefit all gastroenterologists, hematologists, dermatologists, primary care doctors, epidemiologists, physicians who related with community hygiene, as well as basic scien
出版日期Book 2016
關鍵詞H; pylori infection; MALT lymphoma; eradication therapy; gastric carcinoma; gastric ulcer; immune thrombo
版次1
doihttps://doi.org/10.1007/978-4-431-55705-0
isbn_softcover978-4-431-56679-3
isbn_ebook978-4-431-55705-0
copyrightSpringer Japan 2016
The information of publication is updating

書目名稱Helicobacter pylori影響因子(影響力)




書目名稱Helicobacter pylori影響因子(影響力)學科排名




書目名稱Helicobacter pylori網絡公開度




書目名稱Helicobacter pylori網絡公開度學科排名




書目名稱Helicobacter pylori被引頻次




書目名稱Helicobacter pylori被引頻次學科排名




書目名稱Helicobacter pylori年度引用




書目名稱Helicobacter pylori年度引用學科排名




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Human Migrationusing DNA microarray or whole-genome sequencing technology give a broad view on the genome of .. In particular, next-generation sequencers, which can read DNA sequences in less time and at lower costs than Sanger sequencing, enabled us to efficiently investigate not only the evolution of ., but also
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CagArity-regulating kinase PAR1 in a tyrosine phosphorylation-independent manner. We propose a hypothesis that mammalian proteome contains a distinct class of proteins carrying an EPIYA or EPIYA-like sequence, which are imitated by bacterial EPIYA effectors such as CagA to perturb intracellular signalin
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VacA Exhibits Pleiotropic Actions in Host Cellsepithelial cells and LRP1 is involved in the induction of autophagy. Interestingly, it has been suggested that CagA is degraded by VacA-induced autophagy and that the interaction between these two molecules is associated with the pathogenesis of gastric diseases. Therefore, better understanding of t
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Gastric Non-, Helicobacter: Its Significance in Human Gastric Diseasesof the species have been reported. Recent and ongoing prevalence studies indicate a higher clinical relevance of these bacteria than earlier impressions suggested. Current efforts in improving cultivation and detection methodology are contributing to an increased understanding of their microbiology,
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