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Titlebook: Heat Shock Proteins and Inflammation; Willem Eden Book 2003 Springer Basel AG 2003 Activation.Arthritis.bacteria.diseases.immune response.

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發(fā)表于 2025-3-21 17:19:36 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Heat Shock Proteins and Inflammation
編輯Willem Eden
視頻videohttp://file.papertrans.cn/426/425005/425005.mp4
概述Focuses on immunity to HSPs and the impact on the regulation of inflammation.Includes aspects of specific adaptive as well as innate immunity.Gives a basis for understanding the mode of action of HSP-
叢書名稱Progress in Inflammation Research
圖書封面Titlebook: Heat Shock Proteins and Inflammation;  Willem Eden Book 2003 Springer Basel AG 2003 Activation.Arthritis.bacteria.diseases.immune response.
描述Heat shock proteins (HSP) have received ample interest by immunologists over recent years. Initially they were found to be dominantly immunogenic microbial antigens. The connection with inflammation was established when it was uncovered that T cells specific for these antigens have a crucial role in the induction and regulation of experimental arthritis. Since then, the raised presence of immunity to HSPs in virtually all conditions of inflammation, including autoimmune diseases, transplant rejection and atherosclerosis, has emphasised the critical significance of immunity to HSPs in inflammatory diseases.
出版日期Book 2003
關(guān)鍵詞Activation; Arthritis; bacteria; diseases; immune response; immune system; immunity; immunotherapy; inflamma
版次1
doihttps://doi.org/10.1007/978-3-0348-8028-2
isbn_softcover978-3-0348-9410-4
isbn_ebook978-3-0348-8028-2Series ISSN 1422-7746 Series E-ISSN 2296-4525
issn_series 1422-7746
copyrightSpringer Basel AG 2003
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 22:30:17 | 只看該作者
Ad P Koetsolution) was characterized by the building of railways, the beginning of electricity and electrical means of communication such as telegraph, telephone and radio as well as mass production and the production line (e.g. for automobiles).
板凳
發(fā)表于 2025-3-22 04:11:32 | 只看該作者
Heat shock proteins in immune response,nt challenge with live Meth A tumor cells in a classical tumor rejection assay [.-.]. However, immunization of mice with HSP preparations from normal tissues [.] or from antigenically-distinct tumor cells [., .] did not protect the animals against tumor challenge.
地板
發(fā)表于 2025-3-22 07:35:27 | 只看該作者
5#
發(fā)表于 2025-3-22 11:34:57 | 只看該作者
HSP60-peptide interference with CD94/NKG2 receptors,the cell directly through release of lytic granules and/or to produce cytokines. Rapidly accumulating data show that certain MHC Class I specific cell-surface receptors, known previously to critically regulate NK cell functions, appear on large numbers of CD8. T-cells after activation [.-.].
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發(fā)表于 2025-3-22 16:00:27 | 只看該作者
1422-7746 f immunity to HSPs in virtually all conditions of inflammation, including autoimmune diseases, transplant rejection and atherosclerosis, has emphasised the critical significance of immunity to HSPs in inflammatory diseases.978-3-0348-9410-4978-3-0348-8028-2Series ISSN 1422-7746 Series E-ISSN 2296-4525
7#
發(fā)表于 2025-3-22 17:27:43 | 只看該作者
Willem van Eden,Liesbeth Paul,Ruurd van der Zeehinking, which therefore projected a position of weakness, as opposed to strength. It is in such an environment that Alva Myrdal had to operate, being fully cognizant of the many traps along her way, not least those of dominant social norms and perceptions of her time.
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發(fā)表于 2025-3-22 22:11:20 | 只看該作者
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發(fā)表于 2025-3-23 03:01:11 | 只看該作者
Heat shock proteins and experimental arthritis,anding of the disease, several animal models of RA have been developed and have been widely studied. Although these models can be very instructive, no one model reflects all aspects of the human disease illustrating the complexity and heterogeneous nature of RA (Tab. 1).
10#
發(fā)表于 2025-3-23 07:09:33 | 只看該作者
Immunity to heat shock proteins and atherosclerosis,prit-autoantigen. After a brief review of experimental . data and results from animal experiments, we will focus on clinical data confirming our pathophysiological concept that early atherosclerosis is the price we pay for protective immunity against microbial HSP60 or . autoimmunity to eliminate biochemically-altered autologous HSP60.
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