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Titlebook: Handbook of Neurotoxicology; Volume II Edward J. Massaro Book 2002 Springer Science+Business Media New York 2002 Cannabinoid.Ethanol.animal

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書目名稱Handbook of Neurotoxicology
副標(biāo)題Volume II
編輯Edward J. Massaro
視頻videohttp://file.papertrans.cn/422/421775/421775.mp4
概述Includes supplementary material:
圖書封面Titlebook: Handbook of Neurotoxicology; Volume II Edward J. Massaro Book 2002 Springer Science+Business Media New York 2002 Cannabinoid.Ethanol.animal
描述N eurotoxicology is a broad and burgeoning field of research. Its growth in recent years can be related, in part, to increased interest in and concern with the fact that a growing number of anthropogenic agents with neurotoxic potential, including pesticides, 1ead, mercury, and the polytypic byproducts of combustion and industrial production, continue to be spewed into and accumulate in the environment. In addition, there is great interest in natural products, including toxins, as sources of therapeutic agents. Indeed, it is well known that many natural toxins ofbroadly differing structure, produced or accumulated for predatory or defensive purposes, and toxic agents, accumulated incidentalIy by numerous species, function to perturb nervous tissue. Components of some of these toxins have been shown to be useful therapeutic agents and/or research reagents. Unfor- of some neurotoxicants of anthropogenic ori- tunately, the environmental accumulation gin, expecialIy pesticides and metals, has resulted in incidents ofhuman poisoning, some of epidemic proportion, and high levels of morbidity and mortality. Furthermore, an increasing incidence of neurobehavioral disorders, some with baffl
出版日期Book 2002
關(guān)鍵詞Cannabinoid; Ethanol; animal toxin; cocaine; developmental neurotoxicology; drugs of abuse; lead; marijuana
版次1
doihttps://doi.org/10.1007/978-1-59259-165-7
isbn_softcover978-1-61737-194-3
isbn_ebook978-1-59259-165-7
copyrightSpringer Science+Business Media New York 2002
The information of publication is updating

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Developmental Neurotoxicologyrs such as a prolonged course of development, immaturity of the blood-brain barrier (BBB), and lack of regeneration of damaged CNS neurons. One of the more insidious aspects of developmental neurotoxicity is that its functional manifestations may not be readily apparent at birth or in infancy, but r
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Electrophysiologic Evidence of Neural Injury or Adaptation in Cocaine Dependencee QEEG, could be relevant to “staging” the disorder with respect to the identification of reversible vs irreversible components, and could potentially provide an approach to the development or selection of treatment. This chapter focuses on data obtained from a large population of cocaine-dependent
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Dopamine and Its Modulation of Drug-Induced Neuronal DamageIn a broader sense, DA is actually considered the neurotransmitter in the brain’s pleasure center (i.e., the nucleus accumbens) where it decodes the reinforcing effects of diverse stimuli. When the DA neuronal system does not function properly, it is possible that the ensuing disruption in higher-co
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