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Titlebook: Handbook of Copper Pharmacology and Toxicology; Edward J. Massaro Book 2002 Springer Science+Business Media New York 2002 Alzheimer.aquati

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發(fā)表于 2025-3-21 19:45:52 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Handbook of Copper Pharmacology and Toxicology
編輯Edward J. Massaro
視頻videohttp://file.papertrans.cn/422/421103/421103.mp4
概述Includes supplementary material:
圖書封面Titlebook: Handbook of Copper Pharmacology and Toxicology;  Edward J. Massaro Book 2002 Springer Science+Business Media New York 2002 Alzheimer.aquati
描述Edward J. Massaro and a panel of leading biomedical researchers and clinical practitioners review, in-depth, the status of our current knowledge concerning the biochemistry of copper in general and its role in health and disease in particular. Drawing on the wealth of new information emerging from the molecular biology revolution, these experts survey the most important research areas of copper pharmacology and toxicology, including copper proteins and transport, copper toxicity and therapeutics, and copper metabolism and homeostasis. They also discuss the molecular pathogenesis of copper in a variety ofmetabolic diseases, Menkes and Wilson‘s diseases and occipital horn syndrome, as well as the role of copper in Parkinson‘s disease, prion disease, familial amytrophic lateral sclerosis (ALS), and Alzheimer‘s disease.
出版日期Book 2002
關(guān)鍵詞Alzheimer; aquatic organisms; biochemistry; cancer; metabolism; research; toxicity; toxicology
版次1
doihttps://doi.org/10.1007/978-1-59259-288-3
isbn_softcover978-1-61737-266-7
isbn_ebook978-1-59259-288-3
copyrightSpringer Science+Business Media New York 2002
The information of publication is updating

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Suspension and Termination of Construction, humans .. What is so special about this trace element that makes it essential in biology? Copper ions undergo unique chemistry because of their ability to adopt distinct redox states, either oxidized, Cu(II), or reduced, Cu(I). Consequently, Cu ions serve as important catalytic cofactors in redox c
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,The Behavior of Consumers’ Attitudes,rP (prion protein). This molecular transition converts a normal version of the protein (PrP.) into a pathogenic form (PrP.) that constitutes the major component of an unprecedented type of infectious particle (prion) devoid of nucleic acid. Although there is a wealth of information now available abo
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Understanding Consumer Financial Behavior disease (CJD) remains a disease of very low incidence, accounting for 1 in 10. deaths per annum worldwide. BSE and CJD are examples of prion diseases, fatal neurodegenerative conditions .. Before the BSE epidemic, prion diseases were infamous because the cause could not be linked to a known pathoge
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Understanding Contemporary Irelanded for the formation of water molecules are transferred from both sides of the mitochondrial inner membranes in eukaryotic cells and of the cell membrane in prokaryotic cells .. The migration of positive and negative charges from the different sides of the membrane produces electric potential across
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https://doi.org/10.1007/978-1-4842-9309-6een uptake and efflux of copper ions determines the amount of cellular copper. In biological systems, copper is effectively sequestered by binding to molecules such as glutathione or metallothionein or by cytoplasmic transport proteins called copper chaperones .. Consequently, free copper ions are v
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Message Authentication Codes (MACs), in 250,000 ., although in some populations it can be as high as 1 in 40,000 .. A less severe form of the disease, occipital horn syndrome (OHS) or X-cutis laxis, resulting from an allelic variant, has also been described. The clinical symptoms of both Menkes disease and OHS are caused by a lack of
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