Titlebook: Handbook of Atopic Eczema; Thomas Ruzicka,Johannes Ring,Bernhard Przybilla Book 19911st edition Springer-Verlag Berlin Heidelberg 1991 Der

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https://doi.org/10.1007/978-3-663-07330-7ant. In addition, epidemiological studies and case reports mostly do not address the question of the causal relation between the underlying AE and the reported association. Despite these shortcomings, we will attempt to review diseases associated with AE and, if possible, discuss current ideas on ca

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Theorie der Einkommensverteilung,+ T-helper cells, as proven for nickel sensitivity [6]. After sensitization has occurred, any further cutaneous contact with the allergen will provoke the manifestation of an allergic contact eczema [7]. Immunologically, this represents the effector phase of cell-mediated immunity (CMI). Several inv

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https://doi.org/10.1007/978-3-322-88270-7 addition, results were often influenced by cytophilic IgE released from cells bearing Fc receptors for IgE (Fc.R), such as B lymphocytes, monocytes and basophils. Only the development of monoclonal antibody techniques and the rapid progress in molecular biology have facilitated a deeper understandi

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Book 19911st editionattention, not only from the with by authors expert in that particular field, the up-to- medical community, but increasingly from the date knowledge provided is not only extensive but also nonmedical public as well. critical. I believe that this Handbook of Atopic Eczema Since atopic eczema first em

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Minimal Forms of Atopic Eczemansitization to atopic allergens exists [47, 49, 50]. Thus, the diagnosis can only be finally accepted on the basis of a family and personal history, progress monitoring, further clinical features related to other stigmata of the atopic constitution, positive skin reaction (e. g. white dermatographis

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Allergic Contact Eczema and Atopic Eczema+ T-helper cells, as proven for nickel sensitivity [6]. After sensitization has occurred, any further cutaneous contact with the allergen will provoke the manifestation of an allergic contact eczema [7]. Immunologically, this represents the effector phase of cell-mediated immunity (CMI). Several inv

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Regulation of IgE Synthesis addition, results were often influenced by cytophilic IgE released from cells bearing Fc receptors for IgE (Fc.R), such as B lymphocytes, monocytes and basophils. Only the development of monoclonal antibody techniques and the rapid progress in molecular biology have facilitated a deeper understandi