Titlebook: HMG-CoA Reductase Inhibitors; Gerd Schmitz,Michael Torzewski Book 2002 Springer Basel AG 2002 atherosclerosis.cardiovascular.coronary arte

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Sterol-regulatory element binding proteins (SREBPs): gene-regulatory target of statin action,of clinical overt cardiovascular complications, like myocardial infarction or stroke, but has almost no effect on angiographically determined luminal diameter of coronary arteries. These, as well as many morphological and molecular studies, have led to a new paradigm of coronary heart disease, i.e.,

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Cellular effects of HMG-CoA reductase inhibitors on blood cells (monocytes, macrophages, platelets)zed societies. Several important environmental and genetic risk factors are associated with atherosclerosis. Over the past decade, it has become evident that atherosclerosis is not simply an inevitable degenerative consequence of ageing, but rather a chronic inflammatory condition connecting altered

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Pleiotropic effects of HMG-CoA reductase inhibitors on cells of the vascular wall,ts, cholesterol becomes an important risk factor for cardiovascular disease, as demonstrated in clinical trials from the Framingham Heart Study [.–.] and the Multiple Risk Factor Intervention Trial [., .]. Although dietary cholesterol can contribute to changes in serum cholesterol levels, more than

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Cost-effectiveness of statins in primary and secondary prevention of coronary heart disease,e substantial. Consequently, both the primary and secondary prevention of CHD are major objectives of healthcare systems worldwide. Recently, large, multi-centre, clinical trials have demonstrated consistently that these objectives can be achieved though cholesterol-lowering therapy with 3-hydroxy-3

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Milestones in Drug Therapyhttp://image.papertrans.cn/h/image/420250.jpg

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