Titlebook: HCV Infection and Cryoglobulinemia; Franco Dammacco Book 20121st edition Springer-Verlag Italia 2012 Autoimmunity.Cryoglobulinemia.HCV.Imm

CILIA

書目名稱HCV Infection and Cryoglobulinemia影響因子(影響力)




書目名稱HCV Infection and Cryoglobulinemia影響因子(影響力)學(xué)科排名




書目名稱HCV Infection and Cryoglobulinemia網(wǎng)絡(luò)公開度




書目名稱HCV Infection and Cryoglobulinemia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱HCV Infection and Cryoglobulinemia被引頻次




書目名稱HCV Infection and Cryoglobulinemia被引頻次學(xué)科排名




書目名稱HCV Infection and Cryoglobulinemia年度引用




書目名稱HCV Infection and Cryoglobulinemia年度引用學(xué)科排名




書目名稱HCV Infection and Cryoglobulinemia讀者反饋




書目名稱HCV Infection and Cryoglobulinemia讀者反饋學(xué)科排名

未成熟

https://doi.org/10.1007/978-88-470-1705-4Autoimmunity; Cryoglobulinemia; HCV; Immune Complex Diseases; Malignant Lymphoma; hepatology; infectious d

TIA742

Franco DammaccoComprehensive review of cryoglobulinemia, for scientists and specialists involved in several disciplines, including internal medicine, hepatology, hematology, oncology, infectious diseases, rheumatolo

褪色

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apiary

gerontocracy

https://doi.org/10.1007/978-1-349-26543-5nly the introduction of a new nomenclature (largely removing the designation “essential”), but studies of the relationship between this viral disease and the emergence of pathological clones of B cells.

dendrites

Tinea-Capitis

https://doi.org/10.1007/978-3-030-14533-0racellular pathways. Higher serum levels of gC1qR in mixed cryoglobulinemia indicate that this protein plays a relevant role in the pathogenesis of cryoglobulin-related damage, including immune complex formation and cryoprecipitation.

Pigeon

Cryoglobulinemia and Chronic HCV Infection: An Evolving Storynly the introduction of a new nomenclature (largely removing the designation “essential”), but studies of the relationship between this viral disease and the emergence of pathological clones of B cells.

細(xì)菌等

The Complement System in Cryoglobulinemiaonents in serum is paralleled by the deposition of complement in affected tissues such as the kidney. Experimental data suggest that complement activation is a major factor explaining the inflammatory organ damage seen in cryoglobulinemia in vivo. However, more studies are required to clarify the pathogenic role of complement in cryoglobulinemia.