Titlebook: ;

軍械庫(kù)

Lymphoid Tissue Inducer Cells in Intestinal Immunity,both types of lymphoid organs. In the adult, a similar population of cells, termed lymphoid tissue inducer-like cells (LTi-like cells), supports the formation of organized gut-associated lymphoid tissue (GALT) in the intestine, including both isolated lymphoid follicles (ILFs) and cryptopatches (CPs

倔強(qiáng)不能

disrupt

IgA Adaptation to the Presence of Commensal Bacteria in the Intestine,de-ranging impact on host immunity and physiology. One adaptation following intestinal colonisation is increased production and secretion of polyspecific intestinal IgA. In contrast to the strong mucosal immune response to bacterial colonisation, the systemic immune system remains ignorant of these

instill

BRAVE

B Cell Recruitment and Selection in Mouse GALT Germinal Centers,id follicles (ILF) of gut-associated lymphoid tissues (GALT), as a result of continuous B cell stimulation by commensal bacteria. It is generally thought that BCR-mediated antigen recognition controls the recruitment and thus selection of B cells within GALT GCs. However, recent results challenge th

四海為家的人

Structural Insights into Antibody-Mediated Mucosal Immunity,mmune responses are critical for preventing invasion by pathogens. In this chapter, we review structural studies that have illuminated various aspects of mucosal immunity. Crystal structures of IgA1-Fc and IgA-binding fragments of the polymeric immunoglobulin receptor and FcαRI, combined with models

災(zāi)難

小歌劇

arbovirus

Intestinal IgA Synthesis: A Primitive Form of Adaptive Immunity That Regulates Microbial Communitieta. In this chapter we attempt to discuss the mechanisms that underlie this reciprocity and emphasize the key role of mucosal IgA in maintenance of an appropriate segmental distribution of microbiota, which is necessary for immune homeostasis.

Indecisive