Titlebook: ;

培養(yǎng)

,Pathology of Guillain-Barré Syndrome,sis began to appear during the late nineteenth century, long before Guillain, Barré and Strohl’s paper on acute radiculoneuritis with albumino-cytological dissociation was published in 1916. However, the inflammatory changes were regarded as inconstant, late and secondary in an influential review an

RAG

Concrete

,Clinical Features of Guillain-Barré Syndrome,sually a diffuse aching in the back, neck or limbs: it occurred in half of a series of 100 patients (Winer et al. 1988). It may be a prominent feature. Occasionally in children pain may cause meningism and rarely retrocollis. It is sometimes difficult to distinguish between the subsiding pain of the

敘述

Spina-Bifida

,Treatment of Guillain-Barré Syndrome, outpatient setting in patients with minor sensory symptoms and signs. It would be desirable to predict at an early stage which patients were likely to become severely affected and take a long time to recover. Treatment could then be concentrated on this poor prognosis group, leaving patients who we

incontinence

食物

MORPH

,Animal Models of Guillain-Barré Syndrome,to be due to a herpes virus just over 20 years ago (Churchill and Biggs 1967; Nazerian et al. 1968). There are different strains of the causative virus and different inherited susceptibilities of chicken breeds so that infection causes several clinical pictures. These include acute or chronic system

燦爛

Other Inflammatory or Demyelinating Neuropathies, they called “periarteritis nodosa”. Wegener (1936) described the association of vasculitis of small vessels affecting the kidneys and extensive granulomas affecting the sinuses and lungs. By 1947 Klemperer was describing vasculitides, systemic lupus erythematosus and some other disorders which affe

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