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書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes影響因子(影響力)




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes影響因子(影響力)學(xué)科排名




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes網(wǎng)絡(luò)公開度




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes被引頻次




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes被引頻次學(xué)科排名




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes年度引用




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes年度引用學(xué)科排名




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes讀者反饋




書目名稱Growth, Proliferation, and Apoptosis in Hepatocytes讀者反饋學(xué)科排名

壓迫

,Retinoid-Induced Apoptosis in Hepatocellular Carcinoma: A Molecular Basis for “Clonal Deletion” Theresistant to natural retinoic acid. However, acyclic retinoid restored the function of phosphorylated RXR-α and induced its downstream gene, including tissue trans glutaminase, an enzyme that has been implicated in apoptosis. Tissue transglutaminase-dependent apoptosis in HCC cells was independent o

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Growth, Proliferation, and Apoptosis in Hepatocytes978-4-431-67887-8

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Politische Kommunikation in der Online-Weltent liver macrophages) were located in non inflamed areas and were positive for CD 68 but negative for Msi-1. We also observed Msi-1 expression on oval-shaped cells in the inflamed portal area, although further analysis is necessary to determine whether these cells were liver stem cells. The asymmet

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Should Scientists act more Politically?resistant to natural retinoic acid. However, acyclic retinoid restored the function of phosphorylated RXR-α and induced its downstream gene, including tissue trans glutaminase, an enzyme that has been implicated in apoptosis. Tissue transglutaminase-dependent apoptosis in HCC cells was independent o