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書目名稱Growth Hormone and Somatomedins during Lifespan影響因子(影響力)




書目名稱Growth Hormone and Somatomedins during Lifespan影響因子(影響力)學(xué)科排名




書目名稱Growth Hormone and Somatomedins during Lifespan網(wǎng)絡(luò)公開度




書目名稱Growth Hormone and Somatomedins during Lifespan網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Growth Hormone and Somatomedins during Lifespan被引頻次




書目名稱Growth Hormone and Somatomedins during Lifespan被引頻次學(xué)科排名




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書目名稱Growth Hormone and Somatomedins during Lifespan年度引用學(xué)科排名




書目名稱Growth Hormone and Somatomedins during Lifespan讀者反饋




書目名稱Growth Hormone and Somatomedins during Lifespan讀者反饋學(xué)科排名

漸變

BRAWL

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,Vorbedingungen für die Planung, the authors’ laboratory using several transgenic and genetic strains of mice and rats with altered function of the individual axis components. These functional changes all produce profound effects on growth as well as on other components of the hypothalamic-pituitary axis.

monologue

Studies on Peptide 23: A GHRH-Regulated Protein of the Pituitary and Gastrointestinal Tractbe paracrine regulators of GH secretion. However, as pointed out by O’Halloran et al. (1991), a number of conditions must be met to establish a paracrine role for any of the putative regulatory factors. As well the influence may not only be on secretion of GH but also on GH cell proliferation or differentiation.

monologue

Insulin-like Growth Factor Binding Proteins: Biochemical Characterization these proteins, both in their primary structure and resulting from post-translational modification, which contribute to their unique biological functions. Of necessity, the review cannot be comprehensive, but will be illustrated by selected examples, including studies conducted in the author’s laboratory.

Hypomania

Autocrine and Paracrine Effects of Insulin-like Growth Factors and Their Binding Proteinss are devoid of IGF-I receptors (Caro et al. 1988). If the liver is the major producer of IGF-I, the lack of IGF-I mitogenic effect on hepatocytes is a support for the endocrine hypothesis of IGF-I mode of action.

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https://doi.org/10.1007/978-981-13-2242-6ters whose dual effect on GH secretion can be explained by the action of the same molecule either at the level of GHRF- or SRIF-secreting neurons (Fig. 1). This is well exemplified by catecholamines, where α. and α.-β. adrenoceptors exert respectively stimulatory and inhibitory influences on GH secretion (Müller et al. 1991).