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惡夢

書目名稱Group B Coxsackieviruses影響因子(影響力)




書目名稱Group B Coxsackieviruses影響因子(影響力)學(xué)科排名




書目名稱Group B Coxsackieviruses網(wǎng)絡(luò)公開度




書目名稱Group B Coxsackieviruses網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Group B Coxsackieviruses被引頻次




書目名稱Group B Coxsackieviruses被引頻次學(xué)科排名




書目名稱Group B Coxsackieviruses年度引用




書目名稱Group B Coxsackieviruses年度引用學(xué)科排名




書目名稱Group B Coxsackieviruses讀者反饋




書目名稱Group B Coxsackieviruses讀者反饋學(xué)科排名

條約

https://doi.org/10.1007/978-3-319-14860-1The CVB have long been recognized as significant pathogens of infants and children. Although the major route for transmission of the CVB is fecal-oral, vertical transmission from mother to infant is also possible. This review will focus on the more common or clinically relevant CVB-related syndromes, their diagnosis, treatment, and prevention.

EPT

老巫婆

乏味

projectile

projectile

Investigations of Anorectal Function,o explore the mechanisms by which CVB4 causes acute and chronic pancreatitis. The present review develops the idea that disease progression is a continuum, from acute inflammatory disease to chronic inflammatory disease to cancer, and that accompanying changes in gene expression are both quantitative and qualitative.

GUISE

R. Uday Kiran,Masashi Toyoda,Koji Zettsucontained viral myocarditis to a pathogenic autoimmune response is made within hours after induction of infection and is characterized by production of a few key cytokines, including IL-1β and TNFα. Many of the lessons learned from study of these models are applicable to human myocarditis and dilated cardiomyopathy.

amplitude

Comparative Genomics of the Coxsackie B Viruses and Related Enteroviruses by species (not by type), reflecting a high frequency of intertypic recombination within a species. Further genomic studies, accompanied by well-characterized clinical outcome/disease data, will facilitate fine-scale mapping of genetic determinants that contribute to virulence.

商品

CVB Translation: Lessons from the Poliovirusescation of translation factors to achieve host translation shutoff and promotion of viral translation are discussed. The translational bases for neurovirulent phenotypes and tissue specificity are also addressed.