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Titlebook: Glutamate, Cell Death and Memory; Philippe Ascher,Dennis W. Choi,Yves Christen Conference proceedings 1991 Springer-Verlag Berlin Heidelbe

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11#
發(fā)表于 2025-3-23 12:09:42 | 只看該作者
Hauptergebnisse meiner Untersuchungen,and Schwarcz 1976), and stroke (Rothman 1984; Simon et al. 1984). Along with these fonctions, we have demonstrated another role for glutamate: glutamate and related excitatory amino acids may play specific roles in both the generation and degeneration of hippocampal neurons (Mattson et al. 1988a,b, 1989; Mattson and Kater 1989).
12#
發(fā)表于 2025-3-23 16:12:23 | 只看該作者
13#
發(fā)表于 2025-3-23 18:58:51 | 只看該作者
Alterations in Calcium Homeostatic Capacity: A Locus for Constructive and Destructive Neuronal Remoand Schwarcz 1976), and stroke (Rothman 1984; Simon et al. 1984). Along with these fonctions, we have demonstrated another role for glutamate: glutamate and related excitatory amino acids may play specific roles in both the generation and degeneration of hippocampal neurons (Mattson et al. 1988a,b, 1989; Mattson and Kater 1989).
14#
發(fā)表于 2025-3-24 00:24:38 | 只看該作者
In Vitro Neuronal Death: Contrasts Between Excitotoxicity and Chemical Hypoxia, in some way producing the neuronal damage and that, by blocking transmitter release, magnesium was protective. The minimal damage seen in young cultures reflected a lack of synaptic connections early on in culture development.
15#
發(fā)表于 2025-3-24 05:13:09 | 只看該作者
978-3-642-84528-4Springer-Verlag Berlin Heidelberg 1991
16#
發(fā)表于 2025-3-24 09:37:27 | 只看該作者
Glutamate, Cell Death and Memory978-3-642-84526-0Series ISSN 0945-6082 Series E-ISSN 2196-3096
17#
發(fā)表于 2025-3-24 13:56:34 | 只看該作者
18#
發(fā)表于 2025-3-24 16:35:17 | 只看該作者
19#
發(fā)表于 2025-3-24 21:39:05 | 只看該作者
https://doi.org/10.1007/978-3-642-84526-0Alzheimer‘s disease; Alzheimersche Krankheit; Alzheimer′s disease; Cell Death; Glutamat; Glutamate; Zellto
20#
發(fā)表于 2025-3-25 00:56:33 | 只看該作者
The NMDA Receptor: Analogies and Differences with the Non-NMDA Glutamate Receptors and with the Niclutamate receptors coupled to G proteins (metabotropic receptors, see Sladeczek . 1985; Sugiyama . 1987; Nawy and Jahr 1990; Lester . 1990a; Charpak . 1990) and the cloning of a series of cDNA sequences coding for sub-units of the “non-NMDA” receptors which directly gate ion channels (ionotropic rec
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