Titlebook: Glutamate and Addiction; Barbara H. Herman,Jerry Frankenheim,Steven R. Zuki Book 2002 Springer Science+Business Media New York 2002 Opioid

充裕

書目名稱Glutamate and Addiction影響因子(影響力)




書目名稱Glutamate and Addiction影響因子(影響力)學(xué)科排名




書目名稱Glutamate and Addiction網(wǎng)絡(luò)公開度




書目名稱Glutamate and Addiction網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Glutamate and Addiction被引頻次




書目名稱Glutamate and Addiction被引頻次學(xué)科排名




書目名稱Glutamate and Addiction年度引用




書目名稱Glutamate and Addiction年度引用學(xué)科排名




書目名稱Glutamate and Addiction讀者反饋




書目名稱Glutamate and Addiction讀者反饋學(xué)科排名

悠然

ETCH

Glutamate and Neurotoxicitycell death, was proposed to play a role in many pathological conditions, in large part based on the observations that injection of glutamate agonists, notably kainate, could result in neuronal death and biochemical abnormalities resembling the pathology seen in disorders such as Huntington’s disease

BILK

明確

Role of Glutamate and Nitric Oxide in the Acquisition and Expression of Cocaine-Induced Conditioned ization has been the meso-accumbens dopamine system because it has been shown to be involved in mediating the motorie effects of a variety of drugs .. The principal findings do indeed support a role for the mesoaccumbens dopamine projections in the development and expression of behavioral sensitizat

低位的人或事

Interactions of Dopamine, Glutamate, and GABA Systems in Mediating Amphetamine- and Cocaine-Induced ffect and sensitiza-tion, involve, in part, different circuits, even though they undoubtedly share some neuroeffector sys-tems. Starting with these working hypotheses, we initiated a study some 12 years ago that was designed to identify specific neuroeffectors and discrete brain areas that mediate a

低位的人或事

Glutamate Cascade from Metabotropic Glutamate Receptors to Gene Expression in Striatal NeuronsluRs expressed in the . oocyte system, the eight subtypes are currently classified into three functional groups .. Activation of group I mGluRs (mG1uR1/5) increases phosphoinositide (PI) hydrolysis via stimulation of phospholipase C, resulting in the subsequent Ca. release from internal stores and p

enlist

Roles of Glutamate, Nitric Oxide, Oxidative Stress, and Apoptosis in the Neurotoxicity of Methamphetible role of glutamate and of NO in the cascade that leads to methamphetamine (METH)-induced neuro-toxicity. Moreover, this chapter will review briefly recent data that provide conclusive evidence that METH can also cause cell death in various regions of the brain.

Kernel

Nitric Oxide-Dependent Processes in the Action of Psychostimulantsmodulates the release of various neurotransmitters such as dopamine, gultamate, and norepinephrine (.). Thus, there is a reason to consider NO as a major neuromodulator of synaptic transmission in brain.

APNEA