Titlebook: Glucagon III; Pierre J. Lefèbvre Book 1996 Springer-Verlag Berlin Heidelberg 1996 Diabetes.GLP-1.Glukagon.glucagon.molecular biology.patho

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書(shū)目名稱(chēng)Glucagon III影響因子(影響力)




書(shū)目名稱(chēng)Glucagon III影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)Glucagon III網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)Glucagon III網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱(chēng)Glucagon III被引頻次




書(shū)目名稱(chēng)Glucagon III被引頻次學(xué)科排名




書(shū)目名稱(chēng)Glucagon III年度引用




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書(shū)目名稱(chēng)Glucagon III讀者反饋




書(shū)目名稱(chēng)Glucagon III讀者反饋學(xué)科排名

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Learn Blockchain by Building Oneignificant progress in the structural characterization of the glucagon receptor has been made due to the application of affinity-labeling approaches (. et al. 1981; .-. and . 1982; . and . 1984; . and . 1985). This technique represents a useful tool in the gathering of information about minor compon

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ENACT

Glutinous

Learn Data Mining Through Excelctivity (. et al. 1985), but most investigators found the peptide inactive (. 1992). Physiological and pathophysiological studies had clearly shown that the distal intestinal mucosa, in which the glucagon gene is being expressed in the so-called L cells (. et al. 1986; . et al. 1987), contained an i

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https://doi.org/10.1007/978-1-4842-5982-5led “central” epitope) present in all glucagon-containing peptides (including proglucagon itself) and the absence of the C-terminal glucagon epitope, which is masked by the octapeptide in oxyntomodulin and glicentin (Fig. 2). As developed in Sect. B.VII, the “gut GLI” or “enteroglucagon” terminologi

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哺乳動(dòng)物

The Glucagon Receptor Gene: Organization and Tissue Distribution,ignificant progress in the structural characterization of the glucagon receptor has been made due to the application of affinity-labeling approaches (. et al. 1981; .-. and . 1982; . and . 1984; . and . 1985). This technique represents a useful tool in the gathering of information about minor compon

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Glucagon and Glucose Counterregulation,ow the physiological range blood-to-brain glucose transport becomes rate limiting to brain glucose metabolism. Given the survival value of maintenance of the plasma glucose concentration, it is not surprising that physiological mechanisms that very effectively prevent or correct hypoglycemia evolved

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Structure and Function of the Glucagon-Like Peptide-1 Receptor,ved in the stimulation of glucose-induced insulin secretion have now been identified. These are GIP (glucose-dependent insulinotropic polypeptide or gastric inhibitory polypeptide) and GLP-1 (glucagon-like peptide-1).