Titlebook: Glaucoma Update III; Glaucoma Society of G. K. Krieglstein (Direktor der Universit?ts-Augen Conference proceedings 1987Latest edition Spri

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DEI

,Differences in the Amount of ‘Plaque-Material’ in the Outflow System of Eyes with Chronic Simple anterial, which they termed ‘plaques’ or plaque-material. Theses plaques are found in normal eyes of elderly humans as well as in cases of chronic simple glaucoma. Tangential sections through the inner wall and the adjacent cribriform meshwork showed that theses plaques mainly consist of sagittal sect

缺陷

Are Some Retinal Ganglion Cells Killed by Glaucoma Before Others?,ganglion cells are injured first, and what their functional properties are. For example, if we knew that ganglion cells most sensitive to flashing green lights disappear first, then we could design psychophysical tests that exploit early loss of this function. In effect, we could rationally design g

強(qiáng)壯

Distribution of Axonal Transport Injury in the Lamina in Experimental Glaucoma in the Monkey,ocular hypertension. The method used permitted analysis of the distribution of transport block within individual axonal bundles. Experiments were designed to study the earliest detectable injury. Results indicated that transport block occurred first in the posterior lamina along the edges of axonal

syncope

Changes in the Lamina Cribrosa in Experimental Glaucoma in Monkeys,t clear. It needs abundant histological data in every stage, but it is impossible in human. Experimental glaucoma in monkey is not true copy of human, but it can help us to fill up the gap. Recently we succeeded to develope a wedge shaped retinal nerve fiber layer (RNFL) defect with corresponding ro

contrast-medium

Factors Determining the Glaucomatous Optic Nerve Head Damage,m to have “.”: they can have high intraocular pressure (IOP) for many years without any demonstrable optic nerve or visual damage (i.e. ocular hypertension — OHT). Other patients seem to have “.”: they lose vision in a typical glaucomatous fashion even though their IOP is always normal (i.e. low-ten

contrast-medium

Optic Nerve Head Blood Flow is Normal in Chronic Experimental Glaucoma,ppears to be initiated at the scierai lamina cribrosa, just in front of the myelin line in the optic nerve head (Anderson and Hendrickson 1974; Minckler et al. 1978; Quigley and Green 1979; Quigley et al. 1981b). Physical rearrangements of the lamina occur early in the glaucoma damage process in hum

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鴕鳥(niǎo)

Ocular Blood Flow and Visual Loss in Glaucomatous Eyes,s impossible to examine this fundamental problem objectively because of a lack of suitable routine clinical methodologies. I will deal with three aspects; first, the automated instrumentation developed for the routine clinical measurements of ocular hemodynamics and aqueous humor dynamics in individ

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