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Titlebook: Giardia; A Model Organism Hugo D. Luján,Staffan Sv?rd Book 2011 Springer-Verlag Vienna 2011

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發(fā)表于 2025-3-21 18:39:33 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Giardia
副標題A Model Organism
編輯Hugo D. Luján,Staffan Sv?rd
視頻videohttp://file.papertrans.cn/386/385708/385708.mp4
概述Comprehensive volume on Giardia biology and pathogenesis as well as clinical treatment..Molecular biology aspects are described in detail..Separate section for experimental methods in Giardia research
圖書封面Titlebook: Giardia; A Model Organism Hugo D. Luján,Staffan Sv?rd Book 2011 Springer-Verlag Vienna 2011
描述Giardia is a relatively simple eukaryotic microbe, causing acute and chronic diarrhea which has been used as a model to understand complex biological processes occurring in eukaryotic cells. Further, due to its parasitic lifestyle, Giardia is an excellent system for the study of the mechanisms of adaptation and cell differentiation from the perspectives of molecular and cell biology.This book presents a comprehensive review of the current state of knowledge regarding all aspects of Giardia’s biology, including epidemiology, cell and molecular biology, genetics, pathogenesis, diagnostics, and clinical treatment. It was written by internationally renowned authors, the leading researchers in the field including several chapters with techniques and resources available for the study of this microorganism. Questions that need to be addressed to fully understand the molecular mechanisms of the parasite as well as the cause of its pathology are presented. Furthermore, Giardia’s biology is compared with that of other parasites in relation to their complexity.This volume is an indispensable resource for researchers working with this parasite. It is a “must” for libraries and the bookshelves
出版日期Book 2011
版次1
doihttps://doi.org/10.1007/978-3-7091-0198-8
isbn_softcover978-3-7091-1927-3
isbn_ebook978-3-7091-0198-8
copyrightSpringer-Verlag Vienna 2011
The information of publication is updating

書目名稱Giardia影響因子(影響力)




書目名稱Giardia影響因子(影響力)學(xué)科排名




書目名稱Giardia網(wǎng)絡(luò)公開度




書目名稱Giardia網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Giardia被引頻次




書目名稱Giardia被引頻次學(xué)科排名




書目名稱Giardia年度引用




書目名稱Giardia年度引用學(xué)科排名




書目名稱Giardia讀者反饋




書目名稱Giardia讀者反饋學(xué)科排名




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發(fā)表于 2025-3-22 00:07:20 | 只看該作者
Hugo D. Luján,Staffan Sv?rdComprehensive volume on Giardia biology and pathogenesis as well as clinical treatment..Molecular biology aspects are described in detail..Separate section for experimental methods in Giardia research
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Taxonomy of , Speciessame species. This has led to uncertainty in our understanding of the epidemiology of . infections, and particularly the question of host specificity and zoonotic transmission. The lack of morphological characters on which to base a species level taxonomy for the forms of . that infect mammals has n
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Waterborne and Environmentally-Borne Giardiasis community-wide outbreaks. In countries where giardiasis is endemic, the risk of environmentally borne transmission is even greater..In this chapter our knowledge regarding transmission of . these routes is reviewed, with consideration of the relative importance of transmission . these routes, outbr
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in Pets and Farm Animals, and Their Zoonotic Potentialhogen in production and pet animals and the zoonotic potential of animals were prone to more debate. Since long, clinical disease has been associated with giardiasis in companion animals, but over the last few years, an increasing amount of data also confirmed the clinical and subclinical relevance
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Mass Spectrometric Analysis of Phospholipids and Fatty Acids in ,n proposed earlier that this intestinal pathogen lacks the ability to synthesize the majority of its own lipids . and depends on supplies from outside sources. Therefore, the questions as to how this ancient eukaryote utilizes exogenous lipids and synthesizes membranes and organelles are extremely i
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Metabolismacks de novo lipid, de novo purine, and de novo pyrimidine syntheses (relying solely on salvage pathways). . also lacks mitochondria and cytochrome-mediated oxidative phosphorylation and thus trophozoites use glycolysis (from glucose only) and the arginine dihydrolase pathways relying on substrate l
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