Titlebook: Gestational Trophoblastic Disease; Benign to Malignant Bhagyalaxmi Nayak,Uma Singh Book 2021 Springer Nature Singapore Pte Ltd. 2021 Gestat

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Gestational Trophoblastic Disease: An Overview,on of large quantities of hCG is a hallmark. Spontaneous regression is possible—less in Choriocarcinoma. Because of haematogenous spread—it mimics good number of other pathologic conditions. This is the first disseminated solid tumour to be cured by Chemotherapy. Five clinico-pathological forms of GTD are recognised:

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Placental Site and Epithelioid Trophoblastic Tumours: Rare Varieties of Gestational Trophoblastic N of treatment and has a role even in metastatic disease. Though these tumours are relatively chemoresistant, chemotherapy is recommended as adjuvant therapy in select patients with uterine confined disease and in metastatic or recurrent disease. Immunotherapy is being investigated for chemoresistant and recurrent cases.

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Epidemiology of Gestational Trophoblastic Diseases,hoblastic tumor (ETT) are malignant tumors and have varying propensities for local invasion and metastasis. Persistent GTD, also called gestational trophoblastic neoplasia (GTN), includes invasive mole, choriocarcinoma, PSTT, and ETT [1].

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Human Chorionic Gonadotropin,ental hormone secreted after implantation and is commonly detected by urine gravindex test. It interacts with the LHCG receptor of the ovary and maintains the corpus luteum during initial weeks of pregnancy. It is also produced by most of the trophoblastic tumors where the serial quantitative detect

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Molecular Markers in Gestational Trophoblastic Diseases,abortions. Reliable diagnosis based on histomorphologic findings alone is limited and interobserver variability even in a setting of specialty practice is well established. The unique genetic basis of molar gestation enables ancillary testing based on ploidy and paternal contribution. This includes