Titlebook: Genomics-Driven Healthcare; Trends in Disease Pr Yashwant Pathak Book 2018 Springer Nature Singapore Pte Ltd. 2018 Drug delivery.Drug disco

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書目名稱Genomics-Driven Healthcare影響因子(影響力)




書目名稱Genomics-Driven Healthcare影響因子(影響力)學(xué)科排名




書目名稱Genomics-Driven Healthcare網(wǎng)絡(luò)公開度




書目名稱Genomics-Driven Healthcare網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Genomics-Driven Healthcare被引頻次




書目名稱Genomics-Driven Healthcare被引頻次學(xué)科排名




書目名稱Genomics-Driven Healthcare年度引用




書目名稱Genomics-Driven Healthcare年度引用學(xué)科排名




書目名稱Genomics-Driven Healthcare讀者反饋




書目名稱Genomics-Driven Healthcare讀者反饋學(xué)科排名

芭蕾舞女演員

https://doi.org/10.1007/978-3-658-25767-5velopment. In other disease states, research is only just beginning to identify genetic loci that could be responsible for development. This chapter is meant to answer the following question: how much of our health can we control, and what role does genetic variation play in disease development?

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https://doi.org/10.1007/978-3-662-29106-1can vary greatly in their levels of expression, protein folding, membrane localization, and transporter efficiencies [3, 13]. These phenotypic differences can ultimately result in major interindividual variabilities in response to identical drug molecules (i.e., pharmacokinetics (PK), pharmacodynami

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https://doi.org/10.1007/978-3-662-26353-2beling describing risk for adverse drug events, genotype-specific dosing, and/or variations in pharmacokinetic and pharmacodynamic parameters. For a select group of medications, such as codeine and clopidogrel, pharmacogenomic information may even be highlighted in a black box warning further emphas

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https://doi.org/10.1007/978-3-658-25380-6associated with variability in drug response. This review focuses on the genetic polymorphisms of phases I and II DMEs and their associations with drug responses. The drugs discussed in this review requiring a pharmacogenomic test before being prescribed includes efavirenz, voriconazole, clopidogrel

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Die Ausrüstung der textilen Warenal pollutants, where the World Health Organization (WHO) now uses the term “environmental burden of disease” to describe adverse effects of a man-made climate, ecosystem degradation, cumulative rise in pollutants, noise, and electromagnetic fields, etc. While epigenetic environmental triggers can al

Inculcate

Curt Heidermanns,Inge Kirchner-Kühnt these mutations. In this chapter, Alzheimer’s disease (AD), Parkinson’s disease (PD), Huntington’s disease (HD), and amyotrophic lateral sclerosis (ALS) are discussed in detail for their pathophysiology, etiology, and latest symptomatic and preventive treatment. In preventive treatment, the latest