Titlebook: Genomics of Crucifer‘s Host- Pathosystem; Govind Singh Saharan,Naresh K. Mehta,Prabhu Dayal Book 2023 The Editor(s) (if applicable) and T

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,Future Research Priorities of Crucifers’ Host-Pathosystem,n durability of host resistance is required to get better insight on pathogen virulence evolution. There is a need to resolve controversial views on improved resistance by . genes pyramiding and its breakdown. Search of more . genes from wild relatives may provide better resources for some pathogens

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genomics of crucifer’s host-pathosystem. The genomics of plants was initiated after the sequencing of . genome for the first time in 1990. The genome size of . is 125?Mbp containing 25,498 genes encoding proteins from 11,000 families. Now, the genome of all the six . species has been sequenced. The

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Palgrave‘s Dictionary of Political Economys including two mini-chromosomes less than 1?Mb. The fungi causing powdery mildew disease has about four times larger genome size than those of other ascomycetes. The genome of . is 160?Mb in size. The fungi . genome is 57.3?Mb in size with 583 scaffolds. The G:C content is 47.4%. The genome analysi

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https://doi.org/10.1007/978-3-662-53334-5 and genotypes. Phylogenetic analysis and nucleotide sequences have distinguished races of . infecting different crucifers in different countries of the world. . isolates from different crucifers have been characterized based on virulence genes specificity to their hosts in Australia. A single domin

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https://doi.org/10.1007/978-3-662-53342-0toxin Destruxin. The ROS production in .-infected host is very complex. H.O. and OH production is high and low, respectively, in moderately resistant host. Enzymes PPO, POX, and CAT increase in resistant host. Alternaria leaf infection in mustard influences production of carbohydrates (sugars), phen