Titlebook: Genomic Imprinting; An Interdisciplinary Rolf Ohlsson Book 1999 Springer-Verlag Berlin Heidelberg 1999 Activation.biology.cancer.chromosome

injurious

書目名稱Genomic Imprinting影響因子(影響力)




書目名稱Genomic Imprinting影響因子(影響力)學科排名




書目名稱Genomic Imprinting網(wǎng)絡(luò)公開度




書目名稱Genomic Imprinting網(wǎng)絡(luò)公開度學科排名




書目名稱Genomic Imprinting被引頻次




書目名稱Genomic Imprinting被引頻次學科排名




書目名稱Genomic Imprinting年度引用




書目名稱Genomic Imprinting年度引用學科排名




書目名稱Genomic Imprinting讀者反饋




書目名稱Genomic Imprinting讀者反饋學科排名

鄙視

Arterielle Hypertonie bei Diabetes mellituster. The difficulty in studying its role in gene expression is that very few Mendelian mutations cause arrest in epigenetic programming and that chromatin changes occurring many cell divisions before transcription starts are difficult to monitor by biochemical means (Lund et al. 1995a). Given this c

空中

敲竹杠

Postpartales Diabetesscreening,Migeon 1994; Lyon 1996). The result of this is that chromosomally XX females and XY males both effectively have a single dosage of the products of X-linked genes. Thus X-chromosome inactivation fulfils the function of dosage compensation of X-linked genes. In eutherian mammals, typically either one

Flirtatious

Klassifikation des Diabetes mellitus,erefore, to invoke fundamentally novel mechanisms to explain the imprinting phenomenon in mammals. For example, the different factors involved in imprinting, such as the stable propagation of different chromatin states that repress or permit gene transcription, are well-known entities in a wide rang

排名真古怪

排名真古怪

Heart-Rate

Synapse

盡忠

Biguanid-induzierte Laktatazidose,e expression essential for the patterning, morphogenesis and allocation of cellular identities in different parts of the embryo. These patterns are produced by the interaction of multiple transcription factors, repressor and activators present in different parts of the embryo or induced by diffusibl