Titlebook: Genomic Adaptability in Somatic Cell Specialization; Marie A. DiBerardino,Laurence D. Etkin Book 1989 Springer Science+Business Media New

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改革運動

Making Global Partnership Work,changes and the circumstances and mechanisms associated with their occurrence. Stable changes in the structural and functional properties of cells are generally based on modifications at the level of DNA. Five kinds of modifications are known: (1) elimination of DNA sequences; (2) amplification of s

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https://doi.org/10.1007/978-3-319-64510-0ental products, each of these carries a spectrum of identifying markers: distinctive patterns of enzyme activities or unique metabolic products, diagnostic cytoarchitecture or elements of fine structure, and other specificities recognizable microscopically or biochemically. Phenotypic changes in ear

Sinus-Rhythm

,PDA at Work—Exploring Impact in Stability, patterns of protein synthesis. These unique constellations of proteins confer on cells the functional and structural characteristics that enable them to perform their specialized roles in the organism. However, in addition to the overt protein synthetic profile, cells may retain the potential to pr

Sinus-Rhythm

https://doi.org/10.1057/9780230118379elman and Kafatos (1974) to denote the change of the cuticular cells of the moth larval silk gland to those producing HCO. during metamorphosis/development and has since been used in many different contexts. So as not to produce a welter of semantics to replace the term already in use, I shall inste

克制

https://doi.org/10.1057/9781137397881 types acquire a specific cellular phenotype for performing a specialized function, no further changes usually occur. We call this the stability of the differentiated state. It is now widely accepted that the process and stability of cell specialization are under genetic control. But the frequency a

discord

https://doi.org/10.1057/9781137383501d. While some have concluded that cancer cells must arise by the dedifferentiation of normal cells, others consider it more likely that many cancer cells develop from normal cells that never fully differentiated. Since these undifferentiated cancer cells can be shown to share many fundamental charac

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978-1-4615-6822-3Springer Science+Business Media New York 1989