Titlebook: Genome Integrity; Facets and Perspecti Dirk-Henner Lankenau Book 2007 Springer-Verlag Berlin Heidelberg 2007 Chromosom.DNA.DNA Repair.Genet

satisficer

書目名稱Genome Integrity影響因子(影響力)




書目名稱Genome Integrity影響因子(影響力)學科排名




書目名稱Genome Integrity網(wǎng)絡公開度




書目名稱Genome Integrity網(wǎng)絡公開度學科排名




書目名稱Genome Integrity被引頻次




書目名稱Genome Integrity被引頻次學科排名




書目名稱Genome Integrity年度引用




書目名稱Genome Integrity年度引用學科排名




書目名稱Genome Integrity讀者反饋




書目名稱Genome Integrity讀者反饋學科排名

Exhilarate

終點

DNA Polymerases and Diseasesalso critically evaluating those cases where the experimental data are not fully convincing. To this aim, we will first give a?short overview of the three main DNA metabolic events, namely replication, repair and recombination, as well as of the checkpoint pathways acting in response to DNA damage.

BRAVE

虛弱的神經(jīng)

Germline Double-Strand Break Repair and Gene Targeting in ,: A?Trajectory System throughout Evolutio of metazoans. In contrast, most studies concerning DNArepair in mammals focused on somatic cells while only few authors engaged in embryonic stem cells. . represents a?field of in vivo biochemistry systems linking DNArepair research with the power of classical genetics and modern developmental, mol

Presbyopia

Presbyopia

Mechanisms of Non-Homologous DNA End Joining:Aspects of In Vitro Assaysning (NHEJ) is the major pathway of DSB repair.Genetic studies in yeast, human and rodent cell lines displaying increased IR sensitivity and defects inDSB repair have provided insight in the genes involved in NHEJ. These genetic data have been confirmed andcomplemented by in vitro assays which have

懦夫

Role of p53 in Double-Strand Break Repairdouble-strand break repair. In this review, we summarize datafrom biochemical, cell based and in vivo approaches demonstrating that wild-type p53 restrains excessiveand mutagenic DNA exchange events. Thus, p53 appears to exhibit fidelity control of homologous recombination,and recent evidence also s

確定方向

不安