Titlebook: Genetics of Rare Autoimmune Diseases; Javier Martín,Francisco David Carmona Book 2019 Springer Nature Switzerland AG 2019 Genomics.Autoimm

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Studien zur Internationalen Politik immune-related genes, including ., ./., ., and .. Several of these variants have been shown to impact the regulation of their associated genes, playing a role in the incompletely understood etiology of Takayasu arteritis.

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2282-6505 t of autoimmunity.Written by renowned clinicians and researc.The main aim of this volume is to provide an updated overview of the genetic basis of a group of complex disorders that are considered rare individually, but show a relatively high prevalence on the whole in developed countries: the rare a

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Der Naturwissenschaftler als Unternehmerions. More recently, the establishment of international consortia and the use of the novel omic technologies have given a boost to the current knowledge of the pathological mechanisms behind GCA. In this chapter, we will give an updated overview of the recent insights into the genetic basis of GCA.

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,Wesen des Nervenschu?schmerzes,sease heritability. Nevertheless, thanks to the development of large-scale AILDs patient cohorts and the implementation of high-throughput methods such as genome-wide association studies (GWAS), a large number of genetic associations have emerged. In this chapter, we will provide an overview of the recent insights into genetic background of AILDs.

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Giant Cell Arteritis,ions. More recently, the establishment of international consortia and the use of the novel omic technologies have given a boost to the current knowledge of the pathological mechanisms behind GCA. In this chapter, we will give an updated overview of the recent insights into the genetic basis of GCA.

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https://doi.org/10.1007/978-3-540-49695-3ch harbors the strongest known susceptibility factors for this disease. Additionally, we will review the available data in non-HLA regions, highlighting the confirmed risk loci and summarizing those that are only suggested. Finally, we will provide an overview of the main molecular pathways involved in the development of this pathology.