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Titlebook: Genetics of Dyslipidemia; Pascale Benlian Book 2001 Springer Science+Business Media New York 2001 Allele.DNA.Lipid.biosynthesis.cardiovasc

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發(fā)表于 2025-3-21 19:12:42 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Genetics of Dyslipidemia
編輯Pascale Benlian
視頻videohttp://file.papertrans.cn/383/382729/382729.mp4
叢書名稱Basic Science for the Cardiologist
圖書封面Titlebook: Genetics of Dyslipidemia;  Pascale Benlian Book 2001 Springer Science+Business Media New York 2001 Allele.DNA.Lipid.biosynthesis.cardiovasc
描述Profound mortality rates, due to cardiovascular disease, are aworldwide problem. Cardiovascular disease results from complicationsof a silent and chronic arterial disease: atherosclerosis. Thechallenge for the practitioner is adapting diagnostic and therapeuticresponses to prevent this common and complex disease. .Dyslipidemia, are disorders of the metabolism of soluble transportersof lipids in extracellular spaces of the human body (including blood),called lipoproteins. They are major cardiovascular risk factors,causally related with atherosclerosis and are themselvesmultifactorial diseases, resulting from interactions between geneticand environmental factors. .The study of genetic factors has recently taken a new path with thestudy of DNA as an experimental object. More than fifty genes oflipoprotein metabolism have been identified in both theirphysiological actions and their contribution to the pathogenesis ofhuman dyslipidemia. The diversity of observations has refined ourcurrent knowledge of the control of lipid metabolism and energyhomeostasis in living organisms beyond the limits of thecardiovascular system (e.g., brain, immune system, and development).These studies have giv
出版日期Book 2001
關(guān)鍵詞Allele; DNA; Lipid; biosynthesis; cardiovascular; cells; enzymes; gene therapy; genes; genetics; metabolism; pr
版次1
doihttps://doi.org/10.1007/978-1-4615-1517-3
isbn_softcover978-1-4613-5593-9
isbn_ebook978-1-4615-1517-3Series ISSN 1566-0753
issn_series 1566-0753
copyrightSpringer Science+Business Media New York 2001
The information of publication is updating

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Genetics of Dyslipidemia978-1-4615-1517-3Series ISSN 1566-0753
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https://doi.org/10.1007/978-3-642-91371-6cted in the past centuries. The recent emergence and expansion of molecular genetics has given plain and multiple evidence of its reality. Now genes may be used as single objects, dismantled by the direct analysis of their primary sequence on human DNA and grasped by the manipulation of their produc
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Book 2001ion to the pathogenesis ofhuman dyslipidemia. The diversity of observations has refined ourcurrent knowledge of the control of lipid metabolism and energyhomeostasis in living organisms beyond the limits of thecardiovascular system (e.g., brain, immune system, and development).These studies have giv
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1566-0753 ism and energyhomeostasis in living organisms beyond the limits of thecardiovascular system (e.g., brain, immune system, and development).These studies have giv978-1-4613-5593-9978-1-4615-1517-3Series ISSN 1566-0753
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