Titlebook: Genetics of Diabetes Mellitus; William L. Lowe Book 2001 Springer Science+Business Media New York 2001 Diabetes.Diabetes mellitus.genes.ge

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https://doi.org/10.1007/978-3-7091-4803-7upling the electron flux along the respiratory chain to the process of oxidative phosphorylation (OXPHOS). Depending on their energy demand, different cell types contain different amounts of mitochondria, ranging from only a few in the spermatozoon to several thousands in cells of skeletal muscle, h

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https://doi.org/10.1007/978-3-7091-9951-0 basis, most clearly indicated by the roughly 70% concordance in identical sibs, a concordance at least twice as high as in nonidentical sibs, and also considerably higher than the 30-40% concordance observed in identical twins sets, wherein one sib has autoimmune Type 1 diabetes (.). Despite this c

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Der Knigge für das Bankgesch?ft0-cell dysfunction that is manifest as inadequate insulin secretion in the face of insulin resistance and hyperglycemia (see Chapters 7 and 8) (.,.). Previous studies have established a clear genetic predisposition for type 2 diabetes (.–.). The concordance rate among monozygotic twins is 50 to 90%

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Der Kolportagebuchhandel (1869-1905)ons worldwide. The concordance rates for diabetes in monozygotic twins are 50% or higher, whereas those for dizygotic twins are substantially lower, at 10 to 17% (.–.). It is estimated that siblings of diabetic individuals are 1.2 to 3 times more likely to have diabetes compared to the population, d