Titlebook: Genetics and Evolution of Aging; Michael R. Rose,Caleb E. Finch Book 1994 Springer Science+Business Media Dordrecht 1994 Allele.Elongation

gruelling

Mutation-selection balance and the evolution of senescencehese two models can be inferred from the difference between the distribution of mutational effects and the equilibrium distribution of allelic effects of polymorphic loci. There are several experimental designs that, to varying degrees of success, give a quantitative assessment of the distribution o

光明正大

The genetics of aging in the yeast ,lecular mechanisms, and phenotypic features of longevity and senescence, including these: GTP-binding proteins will frequently be involved in determining longevity, asymmetric cell division will be often encountered during embryogenesis while binary fission will be more characteristic of somatic cel

dictator

爆米花

New model systems for studying the evolutionary biology of aging: crustaceasmall body size, and being easily cultured in a laboratory environment. It is also desirable to begin with natural populations that differ in their rate of aging. I present three scenarios and four groups of organisms which fulfill these requirements. The first two scenarios apply to well-documented

gerontocracy

Evolution of aging: testing the theory using ,using experimental manipulations suffers from some of the same problems as measures of genetic correlations; however, these two approaches may be fruitful when used together. The experimental results so far suggest that aging in . has evolved in part as a consequence of selection for an optimal life

AWL

VERT

incite

GLARE

寬敞

The Janiform genetics of aging other face, the population genetics of aging has difficulties with mechanistic content, such that its theories and their tests are often put forward in a manner that resists biological interpretation. If we are to achieve a remotely satisfying scientific understanding of aging, we must bear both faces of the problem in mind.