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Titlebook: Genetic Research in Psychiatry; “2. Münchner Genetik Julien Mendlewicz,Hanns Hippius (Head of Dept. of Conference proceedings 1992 Springe

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書目名稱Genetic Research in Psychiatry
副標(biāo)題“2. Münchner Genetik
編輯Julien Mendlewicz,Hanns Hippius (Head of Dept. of
視頻videohttp://file.papertrans.cn/383/382620/382620.mp4
圖書封面Titlebook: Genetic Research in Psychiatry; “2. Münchner Genetik Julien Mendlewicz,Hanns Hippius (Head of Dept. of  Conference proceedings 1992 Springe
描述Major and exciting advances in psychiatric genetics werediscussed at a 3-day international workshop. Theinternationally renowned editors assembledan impressivelist of specialists, all of whom are leading in theirsubject. Reviews and short articles which stress specialproblems or new research results have been brought togetherin this book, also including intensive discussions of thedifferent topics. Much of thematerial covers theapplication of molecular genetics in majorpsychoses,Alzheimer‘s dementia or preclinical research. However, theproblems of diagnostic features or phenotypicalcharacterizationbroaden further the content of this volume,making it truly a collection of the art information.
出版日期Conference proceedings 1992
關(guān)鍵詞Alzheimersche Krankheit; Molecular Genetics; Molekulargenetik; Psychiatric Genetics; Psychiatrische Gene
版次1
doihttps://doi.org/10.1007/978-3-642-46762-2
isbn_softcover978-3-642-46764-6
isbn_ebook978-3-642-46762-2
copyrightSpringer-Verlag Berlin Heidelberg 1992
The information of publication is updating

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Clinical Observations Critical for Linkage Studies of Schizophrenia and Affective Psychoses,ence of the fact that the mode of inheritance of these disorders is unknown. The design and analysis of linkage trials has been inadequate for psychiatric disorders that are as common and developmentally complex as schizophrenia and affective psychoses. In this paper I will summarize some clinical o
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,Molecular Pathology and Aetiology of Alzheimer’s Disease,s. It accounts for about 60% of the cases of dementia and its prevalence increases logarithmically with age. The clinical manifestations of AD include progressive impairments in short-term and — in later stages — in long-term memory, language, behaviour and disorientation in time and space (McKhann
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,Molecular Genetics of Familial Alzheimer’s Disease, syndrome) using molecular genetic techniques has stimulated several scientists to use a similar approach to Alzheimer’s disease (AD), the major cause of senile dementia in the Western population (Davies 1986).
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