Titlebook: Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2; Barry D. Nelkin Book 1996 Springer-Verlag Berlin Heidelberg 1996 biology.cell.g

obesity

書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2影響因子(影響力)




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2影響因子(影響力)學(xué)科排名




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2網(wǎng)絡(luò)公開度




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2被引頻次




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2被引頻次學(xué)科排名




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2年度引用




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2年度引用學(xué)科排名




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2讀者反饋




書目名稱Genetic Mechanisms in Multiple Endocrine Neoplasia Type 2讀者反饋學(xué)科排名

WAIL

Mutations in Ret in MEN 2,e. This genetic information coupled with a basic understanding of the ways in which the different types of mutation may affect the activity of the ret protein, has already provided interesting insights into the biology of receptor tyrosine kinases. A more detailed elucidation of the genotype: phenot

vitreous-humor

大方一點(diǎn)

切掉

Origin of Adrenal Chromaffin Cells from the Neural Crest,istological techniques based on catecholamine biochemistry allowed early researchers to identify the precursors for adrenal chromaffin cells as they migrate from the primary sympathetic chains into the adrenal primordia. Subsequent production of antisera that recognize catecholamine-synthesizing enz

Liability

Liability

Neuronal Properties of Thyroid C-Cell Tumor Lines,ntrast to the primarily endocrine nature of the parental C-cells. What can account for this transdifferentiation? In this chapter, we will try to answer that question by comparing the neuronal features of C-cell lines with those of normal C-cells treated with nerve growth factor. From that perspecti

Arthropathy

颶風(fēng)

Conclusion,w therapeutic targets for MTC, pheochromocytoma and biologically related cancers. Here, I would like to point out some of the questions which have been raised in the text, and which seem to me to be of special interest for future study. Of course, these choices reflect my bias; and others might emph

不自然

https://doi.org/10.1007/978-3-322-93658-5s has been reviewed extensively by Foulds.. Based on observations of karyotypic progression in hematopoietic and other neoplasms, Nowell. proposed a clonal evolution model for tumor progression. In this model, variant cell clones which possess some growth advantage overgrow the other cells in the tumor.