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Titlebook: Genetic Analysis of the Cell Surface; P. Goodfellow Book 1984 Chapman and Hall 1984 Organelle.Transfusion.chromosome.genetics.growth.molec

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發(fā)表于 2025-3-21 18:07:16 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱(chēng)Genetic Analysis of the Cell Surface
編輯P. Goodfellow
視頻videohttp://file.papertrans.cn/383/382446/382446.mp4
叢書(shū)名稱(chēng)Receptors and Recognition
圖書(shū)封面Titlebook: Genetic Analysis of the Cell Surface;  P. Goodfellow Book 1984 Chapman and Hall 1984 Organelle.Transfusion.chromosome.genetics.growth.molec
描述The cell surface is the barrier between the cell and its environment which regulates the flow of both simple and complex molecules into and out of the cell; it is also the organelle responsible for communication between the cell and its environment. Each cell expresses receptors for a wide variety of hormones, growth factors, growth substrates and other cells. In multicellular organisms communication between cells is required for controlling development, cellular differentiation, morphogenesis and, in a more general sense, integration of myriad cell types into a single organism. The series Receptors and Recognition has as its overall aim the dissection of the cell surface to correlate structure and function for this complex organelle. In most of the preceding volumes the approach has been biochemical or physiological. In this volume the mammalian cell surface is analysed by a genetic approach. Genetic analysis of the cell surface, especially when combined with immuno- logical techniques, has a long history. In 1900 Landsteiner showed that serum from one individual could agglutinate the red cells of another. Besides the practical result of making blood transfusion safe, this was the
出版日期Book 1984
關(guān)鍵詞Organelle; Transfusion; chromosome; genetics; growth; molecular genetics; receptor
版次1
doihttps://doi.org/10.1007/978-94-009-5556-1
isbn_softcover978-94-010-8952-4
isbn_ebook978-94-009-5556-1
copyrightChapman and Hall 1984
The information of publication is updating

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發(fā)表于 2025-3-21 23:56:31 | 只看該作者
Genetics of the Human Red Cell Surface,d to produce antibodies against the red cells of another by cross-reacting environmental antigens (this is the case with ‘naturally occurring’ antibodies to A and B blood groups), pregnancy (e.g. Rhesus antibodies) and accidentally during blood transfusions. All these sources provide a rich supply o
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Analysis of the Human Cell Surface by Somatic Cell Genetics,econd, because of personal choice in mating, laudable ethical constraints on experimenters and small family sizes, matings are frequently less informative than would be the case for experimental animals. Third, the paucity of genetic markers present at polymorphic levels (i.e. greater than 1%) in hu
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Molecular Genetics of the HLA Region,at several books have been devoted to this subject alone (for example, ., Volume 13). Initial interest was spurred by the role of the MHC in transplantation rejection and by the discovery of very high levels of polymorphism at several loci in this region. Genetic studies on the MHC extend from the p
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Cell Genetic Analysis of the Receptor Systems for Bioactive Polypeptides, cell surface. In this latter case the molecules which bind by definition recognize functionally important cell surface molecules. Genetic analysis has been used to map the genes controlling growth factor receptors and to elucidate the mechanism by which growth factors mediate these biological effec
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Genetics of the Cell Surface of the Preimplantation Embryo: Studies on Antigens Determined by Chromnetics and antibodies. A similar approach has been attempted for studying other complex interactions of cells, for example in the early mouse embryo. The starting point for these studies was the theoretical argument that the regulatory products of the MHC might have evolved from a more general syste
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Genetics of the Cell Surface of the Preimplantation Embryo: Studies on Antigens Determined by Chromin males and homozygous lethality at different times in embryogenesis. In 1967, Robert Erickson and Salome Glucksohn-Waelsch proposed that the pleiotropic effects of the . locus could be explained by a series of related genes which controlled development through cell surface molecules which in turn regulated cellular interactions.
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