Titlebook: Gene Regulation by Steroid Hormones IV; Arun K. Roy,James H. Clark Conference proceedings 1989 Springer-Verlag New York, Inc. 1989 DNA.EGF

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https://doi.org/10.1007/978-3-030-66873-0sed the possibility that these growth responses may involve an interplay between the steroid, peptide growth factors, and nuclear protooncogenes. For example, estrogens stimulate secretion of IGF and EGF-like peptides in MCF-7 human breast cancer cells, and these peptides can support MCF-7 cell grow

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DEFER

Tim Hitchcock,Peter King,Pamela Sharped development (Wolff and Wolff, 1969). The active form of the hormone, triiodothyronine (T.), mediates changes in cellular function by binding to limited capacity high affinity receptors located in nuclei of target cells (for review see Oppenheimer et al., 1987). In contrast to steroid hormone actio

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ABASH

Chronik des deutschen Films 1895-1994 steroid receptors and steroid-responsive genes have supported the hypothesis that steroid-receptor complexes affect gene transcription by acting as transcriptional regulators that interact with gene regulatory elements (Yamamoto). Although steroids have dramatic effects on the transcription of cert

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Vereinswesen und Ausstellungen,ammals, it is also responsible for maintaining the sexually dimorphic pattern of steroid metabolism (Roy & Chatterjee, 1983; Gustafsson et al, 1983). A large number of studies investigating the effect of estrogens on the hepatic synthesis of vitellogenins have provided considerable understanding of

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,Aus der forstlichen Ger?thekammer,on of steroid receptors with transcription factors. The cPR A and B proteins differ by an additional 128 amino acids located at the N-terminus of the B protein. These proteins are functionally distinct in terms of their ability to activate specific traget genes. The N-terminal region of the cPR B pr

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Regulation of Glucocorticoid Receptor Protein and Gene Expression by Glucocorticoids,aptational processes of eukaryotic organisms. In general, free circulating glucocorticoids released from the adrenal cortex in response to adrenocorticotropic hormone (ACTH) from the pituitary enter target cells by a passive diffusion process conferred by the high lipid solubility common to all ster