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Titlebook: Gene Regulation by Steroid Hormones II; Arun K. Roy (Professor of Biological Sciences),Jam Conference proceedings 1983 Springer-Verlag New

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書目名稱Gene Regulation by Steroid Hormones II
編輯Arun K. Roy (Professor of Biological Sciences),Jam
視頻videohttp://file.papertrans.cn/382/381952/381952.mp4
圖書封面Titlebook: Gene Regulation by Steroid Hormones II;  Arun K. Roy (Professor of Biological Sciences),Jam Conference proceedings 1983 Springer-Verlag New
描述Receptors and Gene Expression It is now more than three years since the last Meadow Brook Conference on Hormones, and a great deal has happened in the meantime. We have become comfortable with the totally unanticipated fact that the coding sequences of genes are in discontinuous arrangements and that the RNA transcribed from them must be extensively processed to form messenger RNA. We have also learned about the strategy of "mixing and matching" of genetic segments so that a small amount of DNA can go a long way in producing a huge variety of different proteins, as in the immunoglobulin system. The explosive effort directed toward DNA sequence analysis has led us to the conclusion that there are signals within the DNA that specify sites of transcription initiation and possibly sites for interacting with regulatory molecules such as hor- mones and their receptors. The current intense interest in the structure of chromatin beyond the nucleosome-that is, the superstructural characteris- tics of the genetic material-is finally yielding meaningful results that give promise for understanding the regulation of gene activity. ROBERT F. GOLDBERGER Preface Research on the molecular mechanism
出版日期Conference proceedings 1983
關(guān)鍵詞DNA; Endoplasmatisches Reticulum; Hormones; RNA; Steroid; gene expression; proteins; regulation; transcripti
版次1
doihttps://doi.org/10.1007/978-1-4612-5482-9
isbn_softcover978-1-4612-5484-3
isbn_ebook978-1-4612-5482-9
copyrightSpringer-Verlag New York Inc. 1983
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Martine Puylaert M.D., F.I.P.P.there has been much emphasis on the proteolytic degradation of the glucocorticoid receptor into forms that retain the steroid-binding function and either retain or do not retain the DNA-binding site (Wrange and Gustafsson 1978; Sherman et al. 1979). In this paper we review and extend the information
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On the Epidemiology of Heart Failure. The cytosol androgen receptor assays from mouse kidney were recently improved in our laboratory by taking advantage of the stabilizing effect of sodium molybdate on the unoccupied androgen receptors (Wright et al. 1981). In subsequent studies, a reliable method was devised for measurement of nucle
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