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Titlebook: Gasotransmitters: Physiology and Pathophysiology; Anton Hermann,Guzel F. Sitdikova,Thomas M. Weiger Book 2012 Springer-Verlag Berlin Heide

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書目名稱Gasotransmitters: Physiology and Pathophysiology
編輯Anton Hermann,Guzel F. Sitdikova,Thomas M. Weiger
視頻videohttp://file.papertrans.cn/381/380813/380813.mp4
概述Reviews the biological, pharmacological and medical functions of the signalling molecules nitric oxide, carbon monoxide, and hydrogen sulfide.Written by experts.Richly illustrated.Includes supplementa
圖書封面Titlebook: Gasotransmitters: Physiology and Pathophysiology;  Anton Hermann,Guzel F. Sitdikova,Thomas M. Weiger Book 2012 Springer-Verlag Berlin Heide
描述.Since the epochal discovery of the radical and highly toxic gas nitric oxide (NO) as a signaling molecule, two other no less toxic gases – carbon monoxide (CO) and hydrogen sulfide (H.2.S) – have been found to also be involved in a plethora of physiological and pathophysiological functions. The gases termed .gasotransmitters. play an increasingly important role in understanding how signalling into and between cells is modulated and fine-tuned. The advent of gasotransmitters has profoundly changed our way of thinking about biosynthesis, liberation, storage and action mechanisms in cellular signaling. In recent years an impressive amount of new data, distributed throughout the existing literature, has been generated. For this book the editors have recruited distinguished colleagues in the field to summarize and review important biological, pharmacological and medical functions and their implications, as well as methods for the detection of gasotransmitters..
出版日期Book 2012
關(guān)鍵詞BK channels; carbon monoxide; gasotransmitter; hydrogen sulfide; nitric oxide; signaling molecules
版次1
doihttps://doi.org/10.1007/978-3-642-30338-8
isbn_softcover978-3-642-44652-8
isbn_ebook978-3-642-30338-8
copyrightSpringer-Verlag Berlin Heidelberg 2012
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Nitric Oxide: Biological Synthesis and Functions,thelial “e”NOS (or NOS III). In pathophysiology, massive amounts of NOproduced by hyperactive nNOS or highly expressed iNOS can contribute to processes such as neurodegeneration, inflammation, and tissue damage. This chapter will describe principles of NO biosynthesis, regulatory mechanisms controll
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Physiological and Pathophysiological Functions of Hydrogen Sulfide,hase (CBS), an H.S-producing enzyme, are 3 times higher in patients with Down’s syndrome compared to normal individuals. In this article, in addition to the basic properties of H.S, physiological and pathophysiological aspects of this important molecule are reviewed.
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Rekha S. Rajan,Jeanette E. Goddardecreased by cyclic guanosine monophosphate (cGMP) -dependent phosphodiesterases (PDEs) (2 or 3). H.S induced an increase of acetylcholine release and whose effect was mediated by cAMP and Ca.. It is suggested that all three gases are produced at the neuromuscular junction and regulate transmitter re
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