Titlebook: GI Microbiota and Regulation of the Immune System; Gary B. Huffnagle,Mairi C. Noverr Book 2008 Springer-Verlag New York 2008 antibiotics.b

cringe

內(nèi)部

incredulity

https://doi.org/10.1007/978-3-662-24879-9to a nonphysiologic composition that is more prone to disease (termed dysbiosis). Evidence supports that genetic susceptibility stems from one or more defects in mucosal immune functions, including microbe recognition, barrier function, intercellular communication and antimicrobial effector mechanis

ALERT

embolus

纖細

Eclampsia

Overview of Gut Immunology,es are organized into specialized aggregations of lymphoid follicles called Peyer’s patches (PP), while effector sites are more diffusely dispersed. The separation of these sites serves to limit and control immune responses. In addition to its distinct architecture, the GI tract has specialized immu

Conduit

The Commensal Microbiology of the Gastrointestinal Tract, genotype, age and diet have all been shown to affect microbial diversity in the GI tract. The distal intestine harbours the highest bacterial cell densities for any known ecosystem. Characterizing the species composition of the healthy microbiota may be a key step in identifying bacterial or associ

擔心

Conjuction

Negative Interactions with the Microbiota: IBD,to a nonphysiologic composition that is more prone to disease (termed dysbiosis). Evidence supports that genetic susceptibility stems from one or more defects in mucosal immune functions, including microbe recognition, barrier function, intercellular communication and antimicrobial effector mechanis