Titlebook: G Proteins, Receptors, and Disease; Allen M. Spiegel (Chief, Metabolic Diseases Branch Book 1998 Springer Science+Business Media New York

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Haphazard

Albright Hereditary Osteodystrophy, Pseudohypoparathyroidism, and Gs Deficiency, characterized by hypocalcemia, hyperphosphatemia, and elevation of serum PTH in the setting of normal renal function, is the major clinical feature. In the original report by Fuller Albright, PHP patients showed reduced calcemic and phosphaturic responses to injected bovine parathyroid extract comp

opportune

,Gαs-Activating Mutations,ot only in all vertebrates, but also in invertebrates, such as . and .. G. couples receptors for numerous peptide hormones and monoamines to stimulation of adenylyl cyclase. Although it is now clear that G-protein βγ-subunits can independently modulate adenylyl cyclase (positively or negatively depe

arthrodesis

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厚臉皮

,Gα12- and Gα13-Subunits of Heterotrimeric G-Proteins A Novel Family of Oncogenes,receptors in normal and aberrant cell growth. As such, a number of oncogenes have been found to code for altered forms of these receptors ., their ligands (.), or for molecules thought to participate in their growth-promoting pathways .. Another family of structurally related cell-surface receptors

厚臉皮

Hypo- and Hyperthyroidism Caused by Mutations of the TSH Receptor,imarily through a positive control exerted by the pituitary hormone thyroid stimulating hormone (TSH) and a negative control exerted by iodine. In most physiological circumstances the maintenance of eumetabolism requires steady concentrations of circulating thyroid hormones. This is achieved by a cl

parasite

Malcontent

Inactivating and Activating Mutations of the FSH Receptor Gene,that participate in the regulation of reproductive functions, and those of the gonadotropin receptors (R) have received considerable attention. Since the discovery of the first luteinizing hormone receptor (LHR) mutations in 1993 ., more than 15 mutations have been so far reported in this gene (. Ch

誹謗

Nephrogenic Diabetes Insipidus and Vasopressin Receptor Mutations,g tubules contain selective cells known as principal cells, responsive to the neurohypophyseal antidiuretic hormone arginine-vasopressin (AVP). The major action of AVP is to facilitate urinary concentration by allowing water to be transported passively down an osmotic gradient between the tubular fl