Titlebook: Experimental Models of Multiple Sclerosis; Ehud Lavi,Cris S. Constantinescu Book 2005 Springer-Verlag US 2005 Antigen.Chemokine.Nervous Sy

Systole

書目名稱Experimental Models of Multiple Sclerosis影響因子(影響力)




書目名稱Experimental Models of Multiple Sclerosis影響因子(影響力)學(xué)科排名




書目名稱Experimental Models of Multiple Sclerosis網(wǎng)絡(luò)公開(kāi)度




書目名稱Experimental Models of Multiple Sclerosis網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書目名稱Experimental Models of Multiple Sclerosis被引頻次




書目名稱Experimental Models of Multiple Sclerosis被引頻次學(xué)科排名




書目名稱Experimental Models of Multiple Sclerosis年度引用




書目名稱Experimental Models of Multiple Sclerosis年度引用學(xué)科排名




書目名稱Experimental Models of Multiple Sclerosis讀者反饋




書目名稱Experimental Models of Multiple Sclerosis讀者反饋學(xué)科排名

Silent-Ischemia

The Role of Complement in EAE seen in multiple sclerosis. Deposition of complement proteins correlates with areas of demyelination and axonal loss observed in EAE and complement inhibition ameliorates disease. However, the precise mechanisms underlying complement-mediated damage are still largely unknown. The recent use of tran

BAN

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不遵守

Topics in Algebraic Coding Theorycomplex allotypes, which in EAE is in response to induction with certain myelin antigens. Both are complex polygenic diseases where disease traits are controlled by a number of polymorphic genes which individually exhibiting modest effect on disease course. A number of as yet largely unknown genes,

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大廳

surmount

https://doi.org/10.1007/978-3-642-49903-6 in combination with different types of adjuvant. The second approach of inducing EAE consists in adoptive transfer of activated T cells that are specific for myelin-associated autoantigens of the central nervous system (CNS). This chapter presents an overview of some useful strategies and protocols

Osteoporosis

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