找回密碼
 To register

QQ登錄

只需一步,快速開(kāi)始

掃一掃,訪問(wèn)微社區(qū)

打印 上一主題 下一主題

Titlebook: Experimental Models of Chronic Inflammatory Diseases; Leonard Eleazar Glynn,Horst Dieter Schlumberger Conference proceedings 1977 Springer

[復(fù)制鏈接]
查看: 37187|回復(fù): 52
樓主
發(fā)表于 2025-3-21 18:37:23 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases
編輯Leonard Eleazar Glynn,Horst Dieter Schlumberger
視頻videohttp://file.papertrans.cn/319/318882/318882.mp4
叢書(shū)名稱Bayer-Symposium
圖書(shū)封面Titlebook: Experimental Models of Chronic Inflammatory Diseases;  Leonard Eleazar Glynn,Horst Dieter Schlumberger Conference proceedings 1977 Springer
描述With the introduction of antibiotics acute inflammatory disease has ceased to be the dominant problem in general medical practice and its place is now increasingly occupied by chronic inflamma- tory disease of which the rheumatic diseases constitute the most important group. lwo aspects of these diseases need to be consid- ered, their aetiology and their pathogenesis. In some respects the latter is more important since even when the aetiology is known, as for example the haemolytic streptococcus in rheumatic fever, the mechanism by which the infecting agent accomplishes the development of the lesions that characterise the disease are still largely unknown. Still more so is this the case in rheuma- toid arthritis and other chronic inflammations where the aetio- logical agents are unknown. In an attempt to clarify the pathogenic mechanisms involved, sev- eral attempts have been made to induce comparable lesions in ex- perimental animals, partly to test underlying hypotheses, and partly to provide test situations for the trial of new therapeu- tic agents. In view of the deficiencies in the current drug treat- ment of chronic inflammatory disease there are many who feel that this is la
出版日期Conference proceedings 1977
關(guān)鍵詞Arthritis; antibiotics; diseases; inflammation; pathogenesis; rheumatic diseases; rheumatism
版次1
doihttps://doi.org/10.1007/978-3-642-66573-8
isbn_softcover978-3-642-66575-2
isbn_ebook978-3-642-66573-8Series ISSN 0067-4672
issn_series 0067-4672
copyrightSpringer-Verlag Berlin Heidelberg 1977
The information of publication is updating

書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases影響因子(影響力)




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases影響因子(影響力)學(xué)科排名




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases被引頻次




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases被引頻次學(xué)科排名




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases年度引用




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases年度引用學(xué)科排名




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases讀者反饋




書(shū)目名稱Experimental Models of Chronic Inflammatory Diseases讀者反饋學(xué)科排名




單選投票, 共有 0 人參與投票
 

0票 0%

Perfect with Aesthetics

 

0票 0%

Better Implies Difficulty

 

0票 0%

Good and Satisfactory

 

0票 0%

Adverse Performance

 

0票 0%

Disdainful Garbage

您所在的用戶組沒(méi)有投票權(quán)限
沙發(fā)
發(fā)表于 2025-3-21 20:40:41 | 只看該作者
Persistence of Antigen in Experimental Allergic Monoarthritismmon with rheumatoid arthritis: it is chronic, it is immunologically induced and the histological picture of the synovitis is similar to that seen in rheumatoid arthritis (1, 2). In this experimental model it has been previously shown in our laboratory that antigen and antibody, in the form of immun
板凳
發(fā)表于 2025-3-22 02:59:05 | 只看該作者
Experimental Subcutaneous Granulomata Simulating R. A. Nodulesand histiocytes. The necrotic area contains collagen fibres in various stages of fragmentation and dissolution, intimately associated with fibrin and other serum proteins. The cells bordering the necrotic zone are characteristically elongated at right angles to its surface in the form of a palisade.
地板
發(fā)表于 2025-3-22 06:48:37 | 只看該作者
5#
發(fā)表于 2025-3-22 10:58:08 | 只看該作者
Rheumatoid-Type Lymph Node Changes in Animals Exhibiting Immunoregulatory Defectslogic studies have revealed marked hypertrophy of the B-cell microenvironments (6) with excessive germinal centre formation and plasma cell cytodifferentiation, both in organised lymphoid tissue (7, 19, 22) and at ectopic foci (12, 21, 27). The hyperplastic changes in lymph nodes may, at times, be s
6#
發(fā)表于 2025-3-22 14:03:13 | 只看該作者
7#
發(fā)表于 2025-3-22 18:05:04 | 只看該作者
8#
發(fā)表于 2025-3-23 00:13:28 | 只看該作者
Viral Replication in Lymphocytes and the Pathogenesis of Connective Tissue Diseasesis are caused by viruses. The reasons for still pursuing this possibility are threefold. Firstly, the clinical features of those diseases are entirely compatible with persistent virus infections; secondly, proven virus infections have not uncommonly provoked disorders closely resembling these diseas
9#
發(fā)表于 2025-3-23 05:21:36 | 只看該作者
10#
發(fā)表于 2025-3-23 09:15:06 | 只看該作者
Antibody Affinity, Macrophage Function and Immunoregulation in Murine Immune Complex Diseasetigens has shown that both quantitative and qualitative variations in antibody response underlie differences in susceptibility to this disease (3, 25). Furthermore, the observations that there are clear inter-strain differences in susceptibility to chronic ICD following neonatal infection of inbred
 關(guān)于派博傳思  派博傳思旗下網(wǎng)站  友情鏈接
派博傳思介紹 公司地理位置 論文服務(wù)流程 影響因子官網(wǎng) 吾愛(ài)論文網(wǎng) 大講堂 北京大學(xué) Oxford Uni. Harvard Uni.
發(fā)展歷史沿革 期刊點(diǎn)評(píng) 投稿經(jīng)驗(yàn)總結(jié) SCIENCEGARD IMPACTFACTOR 派博系數(shù) 清華大學(xué) Yale Uni. Stanford Uni.
QQ|Archiver|手機(jī)版|小黑屋| 派博傳思國(guó)際 ( 京公網(wǎng)安備110108008328) GMT+8, 2025-10-14 20:55
Copyright © 2001-2015 派博傳思   京公網(wǎng)安備110108008328 版權(quán)所有 All rights reserved
快速回復(fù) 返回頂部 返回列表
东乌珠穆沁旗| 成安县| 凤山市| 巴彦淖尔市| 磐安县| 肥城市| 大同县| 通化县| 怀集县| 丽江市| 西和县| 民丰县| 醴陵市| 泗水县| 酒泉市| 中山市| 怀宁县| 南开区| 太白县| 广宁县| 油尖旺区| 涪陵区| 阳原县| 大洼县| 扎赉特旗| 渭源县| 安阳县| 科技| 资溪县| 班戈县| 克山县| 施秉县| 永年县| 绍兴市| 沂水县| 平度市| 桓台县| 汉沽区| 简阳市| 贵定县| 蒲城县|