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Titlebook: Experimental Models of Cardiovascular Diseases; Methods and Protocol Kiyotake Ishikawa Book 2024Latest edition The Editor(s) (if applicable

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發(fā)表于 2025-3-25 05:56:22 | 只看該作者
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發(fā)表于 2025-3-25 07:42:17 | 只看該作者
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發(fā)表于 2025-3-25 13:31:12 | 只看該作者
Elias C. Tonias,Constantine N. Tonias disease. Induction of reproducible infarct size is crucial for quantitative and analytical purpose. Here we describe a quick and reliable method to induce consistent infarct size in mice in less than a minute.
24#
發(fā)表于 2025-3-25 18:21:18 | 只看該作者
Eric Ballot,Pierre Bourdet,Fran?ois Thiébautd the complex pathophysiology and mechanisms that lie behind this disease setting, and design/evaluate appropriate treatment strategies, suitable animal models are required. Described here are the protocols for establishing surgically induced animal models of CRS including important methods to deter
25#
發(fā)表于 2025-3-25 20:33:55 | 只看該作者
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發(fā)表于 2025-3-26 02:35:40 | 只看該作者
Geometric Properties for Incomplete Dataeling, and the development of concentric laminar intimal fibrosis with plexiform lesions. While rodent models have been developed to study PH, they have certain deficiencies and do not entirely replicate the human disease due to the heterogeneity of PH pathology. Therefore, combined models are neces
27#
發(fā)表于 2025-3-26 05:03:09 | 只看該作者
Silvia Cingolani,Kazunaga Tanakaing the extent of damage and better managing heart failure progression are urgently needed. Preclinical large animal models are a critical step in the translation of scientific discoveries toward clinical trials and therapeutic application. In this chapter, we detail methods to induce swine models o
28#
發(fā)表于 2025-3-26 10:18:12 | 只看該作者
29#
發(fā)表于 2025-3-26 16:08:20 | 只看該作者
https://doi.org/10.1007/978-1-0716-3846-0hemodynamics; valvular dysfunction; cardiomyocytes; pulmonary hypertension; ischemia-reperfusion injury
30#
發(fā)表于 2025-3-26 17:23:23 | 只看該作者
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