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Titlebook: Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond; Bernward Garthoff,Andreas M. Knorr,Friedel Seuter Conference

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發(fā)表于 2025-3-21 16:52:57 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond
編輯Bernward Garthoff,Andreas M. Knorr,Friedel Seuter
視頻videohttp://file.papertrans.cn/319/318845/318845.mp4
圖書封面Titlebook: Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond; Bernward Garthoff,Andreas M. Knorr,Friedel Seuter Conference
描述At the workshop held in Leverkusen on December 1, 1993, Professor Dr. Stanislav Kazda was honored on the occasion of his retirement for his scientific contributions and his involvement in drug development in our company, Bayer AG. In a recent article by Eugene Garfield (Current ContentslLife Sciences 36:3-15, 1993), what most of us participating in this workshop have supposed for years was confirmed: that Kazda is one of the leading scientific authors in academia worldwide. In the said survey of citations of biomedical research articles, Kazda is number 29 in terms of actual impact, individual ~itations, and total number of published manuscripts (and this only covers the time period from 1981 to 1992). In addition, his work has contributed significantly to the number eight ranking of our company in the so-called Corporation List. With the support of colleagues inside and outside the company, Kazda made a new substance class, the 1, 4-dihydropyridines, discovered by Bossert and Vater, well known to the scientific community on a worldwide basis and established its outstanding usefulness in hypertension. He has contributed numerous publi- cations on the pharmacology of these calcium a
出版日期Conference proceedings 1995
關(guān)鍵詞Calcium antagonists; Calciumantagonisten; Hochdruck; Pharmakologie; Stroke; hypertension; pathophysiology;
版次1
doihttps://doi.org/10.1007/978-3-642-79338-7
isbn_softcover978-3-540-58545-9
isbn_ebook978-3-642-79338-7
copyrightSpringer-Verlag Berlin Heidelberg 1995
The information of publication is updating

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沙發(fā)
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Erich Grie?ler,Harald Rohracher first argument for the implication of a change in Ca. Channels in hypertension is the blood pressure-lowering effect of dihydropyridine Ca. antagonists (MacGregor et al. 1982; Kazda and Knorr 1990). These antagonists have also been shown to suppress the myogenic active tone displayed by blood vesse
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Pharmacogenetic Study in Asthma,histicated preparation far from the physiological State. Therefore, we decided to study this question in anesthetized dogs in which the CA were infused directly into the left renal artery (LRA) at a dose not influencing systemic blood pressure [8]. Nevertheless, the . effects of CA are clinically no
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Functional Modifications in Blood Vessels of Hypertensive Rats, first argument for the implication of a change in Ca. Channels in hypertension is the blood pressure-lowering effect of dihydropyridine Ca. antagonists (MacGregor et al. 1982; Kazda and Knorr 1990). These antagonists have also been shown to suppress the myogenic active tone displayed by blood vesse
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發(fā)表于 2025-3-22 13:22:54 | 只看該作者
Influence of Calcium Antagonists on Kidney Function of Hypertensive Rats,histicated preparation far from the physiological State. Therefore, we decided to study this question in anesthetized dogs in which the CA were infused directly into the left renal artery (LRA) at a dose not influencing systemic blood pressure [8]. Nevertheless, the . effects of CA are clinically no
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Experimental Hypertension and Therapeutic Progress: Vasodilation and Beyond978-3-642-79338-7
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Protection Against Hypertensive Tissue Damage,anda of the pharmaceutical industry. In my experience, this sceptical attitude is rather common among critical clinicians. And frankly speaking, from their point of view, such an attitude is fairly well substantiated.
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