Titlebook: Exercise, Calories, Fat and Cancer; Maryce M. Jacobs Book 1992 The Editor(s) (if applicable) and The Author(s), under exclusive license to

GENUS

類似思想

https://doi.org/10.1007/978-3-030-56743-9cies toward development of coronary heart disease, a chief cause of early debilitation and premature death. But the evidence that physical activity influences cancer incidence is less clear, perhaps because cancer is a variety of diseases differing in underlying cause, age of onset, organ of involve

NEEDY

獸群

侵略

https://doi.org/10.1007/978-1-4614-0637-2tivity being promoted for its health benefits. The influence of exercise on the induction of breast cancer in an animal model for the disease was investigated. The data presented indicate that both the intensity and duration of exercise affect the promotion stage of chemically induced mammary carcin

Congeal

https://doi.org/10.1057/978-1-137-59721-2hronic diseases, with their long latencies, appear to be especially susceptible to the influence of diet. Diet affects the pathogenesis of coronary heart disease, gallstones, appendicitis, varicose veins, obesity, hiatus hernia, and cancer.. Indeed, in the last case, it has been suggested that at le

整理

profligate

https://doi.org/10.1007/978-3-8349-8462-3rtain types of cancers, one of the most frequently recommended dietary changes is to lessen the percentage of calories provided by fat. The question that will be addressed herein is: What is the evidence for a . role for dietary fat as a factor determining the incidence of cancer in laboratory anima

Capitulate

,Zur Konkurrenz der Dreiecksh?hen,owing body of evidence implies that this disease may be preventable to a significant degree. Epidemiologic evidence. indicates that populations consuming diets high in fat are at greater risk for colon cancer than are populations consuming diets low in fat. Concordance for these findings has been ob

Amenable