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Titlebook: Etiology of Cancer in Man; Arthur S. Levine Book 1989 Kluwer Academic Publishers 1989 AIDS.cancer.carcinoma.hormones.leukemia.liver.mesoth

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發(fā)表于 2025-3-21 17:04:47 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Etiology of Cancer in Man
編輯Arthur S. Levine
視頻videohttp://file.papertrans.cn/317/316392/316392.mp4
叢書名稱Cancer Growth and Progression
圖書封面Titlebook: Etiology of Cancer in Man;  Arthur S. Levine Book 1989 Kluwer Academic Publishers 1989 AIDS.cancer.carcinoma.hormones.leukemia.liver.mesoth
描述This volume reviews our current knowledge concerning can- Several chapters discuss the contributions of genetic asp- cer growth and progression as it relates to the etiology of ects, metabolism, endocrine-related aspects and nutrition to human cancer. As emphasized in Volumes I-V of this series, cancer progression. Moreover, our current knowledge con- neoplastic diseases are multistep maladies. There are many cerning urbanization factors, radiation, therapy-induced causes for the appearance of neoplastic diseases. Earlier neoplasms, environmentally induced neoplasms (e. g. , chapters in the series have reviewed molecular and cellular mesotheliomas induced by asbestos) and malignant neo- aspects of tumor initiation, promotion and progression to plasms in organ transplant recipients are summarized. the invasive and metastatic phenotype. Contributions to the The impact of AIDS on neoplasm development is re- initiation and progression of neoplastic diseases are made by viewed from an epidemiologic perspective that explores mul- natural features of the environment and by its contaminants tiple facets of immunity, infectious disease, sexual behavior and by nutritional factors. Neoplastic
出版日期Book 1989
關(guān)鍵詞AIDS; cancer; carcinoma; hormones; leukemia; liver; mesothelioma; pregnancy; tumor; tumorigenesis
版次1
doihttps://doi.org/10.1007/978-94-009-2532-8
isbn_softcover978-94-010-7644-9
isbn_ebook978-94-009-2532-8Series ISSN 1871-3238
issn_series 1871-3238
copyrightKluwer Academic Publishers 1989
The information of publication is updating

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Relationships Between Malignant and Non-Malignant Disease of Western Culture,r approach. Such observations are often relatively inexpensive and have often constituted the stones with which hypotheses have been erected. These can then be tested experimentally and subsequently discarded, modified or confirmed.
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Genetic Diseases, Hamartomas, and Familial Occurrence of Neoplasms,r resistance governs responses of individuals to these agents. This interaction between environmental carcinogens and genetic factors has been termed ecogenetics. Ultimately, the final step in the genesis of malignant neoplasms involves molecular and/or cytogenetic alterations that free the cell fro
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Selected Aspects of Primary Liver Carcinoma in Japan,langioma’ that had been commonly used worldwide until recently, were coined by Prof. Yamagiwa in 1911 (28), who also pioneered in experimental carcinogenesis by producing carcinoma on the ear of a rabbit after repeated application of coal tar. In 1932, Prof. Yoshida for the first time succeeded in p
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Effects of Localized and Disseminated Cancers on Metabolism and Nutrition in Man, diagnosis have decreased mean survival compared to similar cancer patients without weight loss. Provision of excess calories alone does not appear to change median survival in patients with advanced cancer and many patients either maintain body weight or lose weight while receiving calories which w
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Assessment of the Endocrine Risk of Developing Breast Cancer,certain diseases, such as breast cancer and high blood pressure, among others. For such risk assessment, a small number of selected clinically healthy women of three age groups was extensively sampled in two geographic locations. Data on twelve plasma hormones in addition to those on some systemic v
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