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Titlebook: Etiology and Morphogenesis of Congenital Heart Disease; From Gene Function a Toshio Nakanishi,Roger R. Markwald,Hiroyuki Yamagi Book‘‘‘‘‘‘‘

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發(fā)表于 2025-3-21 19:35:48 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
書目名稱Etiology and Morphogenesis of Congenital Heart Disease
副標(biāo)題From Gene Function a
編輯Toshio Nakanishi,Roger R. Markwald,Hiroyuki Yamagi
視頻videohttp://file.papertrans.cn/317/316390/316390.mp4
概述Focuses on the etiology and morphogenesis of congenital heart disease.Includes contributions by an international group of distinguished scientists and clinicians.Highlights the latest research topics
圖書封面Titlebook: Etiology and Morphogenesis of Congenital Heart Disease; From Gene Function a Toshio Nakanishi,Roger R. Markwald,Hiroyuki Yamagi Book‘‘‘‘‘‘‘
描述.This volume focuses on the etiology and morphogenesis of congenitalheart diseases. It reviews in detail the early development and differentiation ofthe heart, and later morphologic events of the cardiovascular system, coveringa wide range of topics such as gene functions, growth factors, transcriptionfactors and cellular interactions that are implicated in cardiac morphogenesisand congenital heart disease. This book also presents recent advances in stemcell and cell sheet tissue engineering technologies which have the potential toprovide novel in vitro disease models and to generate regenerative paradigmsfor cardiac repair and regeneration. ??Thisis the ideal resource for physician scientists and investigators looking forupdates on recent investigations on the origins of congenital heart disease andpotential future therapies..
出版日期Book‘‘‘‘‘‘‘‘ 2016
關(guān)鍵詞Cellular Interaction; Congenital Heart Disease; iPS cells; Gene functions; Growth factors; Transcription
版次1
doihttps://doi.org/10.1007/978-4-431-54628-3
isbn_softcover978-4-431-56633-5
isbn_ebook978-4-431-54628-3
copyrightThe Editor(s) (if applicable) and the Author(s) 2016
The information of publication is updating

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Working with/in Deleuzo-Guattarian Ideas,ription factors and small molecules that reprogram human fibroblasts into cardiomyocyte-like cells and are testing these in large animals. Knowledge regarding the early steps of cardiac differentiation in vivo has led to effective strategies to generate necessary cardiac cell types for regenerative
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Working with Photography Projects,different mechanisms of glucose and lactate metabolism. Based on these findings, we succeeded in metabolically selecting cardiomyocytes using glucose-free and lactate-supplemented medium, with up to 99?% purity and no teratoma formation. Using our aggregation technique, we also showed that >90?% of
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Developing Property Rights Theoryto override biochemical or structural deficits of CHD associated with valve and septal signaling hubs or fibroblast/myocyte interactions. Preliminary tests of the model indicate remedial potential for cardiac injuries.
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Dangeni,Rui He,Natthaphon Tripornchaisakunborn human fetal population. Clinically, congenital heart disease patients with ciliary dysfunction were found to have more respiratory symptoms and disease, a finding with significant clinical implications, as congenital heart disease patients undergoing surgical palliation often have respiratory
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https://doi.org/10.1007/978-1-4302-0802-0ontal septum and, in the other taxa studied, into the folding part of the interventricular septum. The vertical septum, indistinct in most reptiles, arises in crocodilians and pythonids at the posterior ventricular wall. It is homologous to the inlet septum in mammals and birds. Eventually, the vari
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The Arterial Epicardium: A Developmental Approach to Cardiac Disease and Repaires dormant, losing the activity of many genes..In human cardiac diseases, both arterial and venous epicardium can be activated again into EPDCs. The epicardial reactivation observed after experimental myocardial infarction and during aneurysm formation of the ascending aorta provides clinical releva
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