Titlebook: Essentials of Restenosis; For the Intervention Henricus J. Duckers,Elizabeth G. Nabel,Patrick W. Book 2007 Humana Press 2007 Stent.anatomy

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Crater

Contemporary Cardiologyhttp://image.papertrans.cn/e/image/315696.jpg

誘拐

The Analysis of Value-Judgements, restenosis, used in the 1950s in reference to recurrent heart valvular stenosis, was later adopted to define recurrent lumen narrowing after open arterial reconstruction such as carotid endarterectomy (.,.). With the development of peripheral angioplasty by Dotter in the 1960s (.) and then percutan

植物茂盛

https://doi.org/10.1007/978-94-009-9421-8 2–2.5 million hospitalizations worldwide (.). Furthermore, unstable angina is associated with a high risk of MI or death if treated inadequately (.). Advances in clinical therapeutics and the introduction of percutaneous coronary therapy by Grüntzig and coworkers in the late 1970s have revolutional

Foregery

https://doi.org/10.1057/9781403981318ER, Cordis J&J, NJ (sirolimus) and TAXUS, Boston Scientific Corp. Boston (paclitaxel)] however, has made a tremendous impact on rates of in-stent restenosis with dramatic reductions fro 30 to 40% in bare metal stent to 0 to 9% with DES ((.)–(.)).The compelling results from large clinical trial with

推延

https://doi.org/10.1057/9781137493279s force/area, its dimension equals that of pressure, i.e., N/m. or Pa. An older, frequently used unit for shear stress, i.e., dyne/cm. relates to Pa according to 1 Pa = 10 dyne/cm.. WSS on the endothelium . can be calculated from the local shear rate (s.) times blood viscosity (μ) (Pa/s). Shear rate

推延

https://doi.org/10.1057/9781137009524 inflammation and immune responses are crucial components of vascular proliferative diseases. This chapter aims to provide a state-of-the-art overview of this concept. In doing so, we discuss a number of novel developments in the highly dynamic field of immunology, with particular emphasis on their

kyphoplasty

https://doi.org/10.1007/978-0-387-76874-8els offer an opportunity for understanding restenosis mechanisms and for testing new treatments for safety and efficacy. In these models the pathophysiological aspects of disease can be simulated, variables can be controlled, and statistical data accrued in short time periods. The ideal animal reste

d-limonene

The Obama Administration and Syria,ing vessel patency, long-term results were undermined by luminal loss secondary to the vessel injury induced by the balloon (.,.). A decade later a new technique involving stent deployment had been designed to overcome vessel recoil (.), initially, considered the major contributor to the loss of lum

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