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Titlebook: Epigenetics, Energy Balance, and Cancer; Nathan A. Berger Book 2016 Springer International Publishing Switzerland 2016 Diabetes.Epigenome.

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發(fā)表于 2025-3-21 18:15:43 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Epigenetics, Energy Balance, and Cancer
編輯Nathan A. Berger
視頻videohttp://file.papertrans.cn/314/313250/313250.mp4
概述Deftly connects the state-of-the-art research in epigenetics and energy balance with cancer.Authoritatively explains the processes involved in epigenetic modifications of DNA, which aspects of energy
叢書名稱Energy Balance and Cancer
圖書封面Titlebook: Epigenetics, Energy Balance, and Cancer;  Nathan A. Berger Book 2016 Springer International Publishing Switzerland 2016 Diabetes.Epigenome.
描述This volume of .Energy Balance and Cancer. provides state-of-the-art descriptions of the rapidly evolving science of epigenetics and how it may explain mechanisms by which alterations in energy balance such as obesity and physical activity may impact cancer. In addition to chapters explaining the processes mediating epigenetic regulation, the volume contains a series of chapters explaining how environmental influences including early life events, nutrition, exercise and microbiota may induce epigenetic changes that can affect carcinogenesis. The following chapters describe epigenetic relations of energy balance to cancer in distinct organ systems including esophagus, colon, prostate and breast. .Epigenetics, Energy Balance and Cancer. provides a valuable resource for students, research investigators and clinicians seeking to better understand these processes as well as a basis for novel translational and transdisciplinary approaches to further elucidate these processes and develop preventive and therapeutic strategies.
出版日期Book 2016
關(guān)鍵詞Diabetes; Epigenome; Nutrition; Obesity; Prostate
版次1
doihttps://doi.org/10.1007/978-3-319-41610-6
isbn_softcover978-3-319-82395-9
isbn_ebook978-3-319-41610-6Series ISSN 2199-2622 Series E-ISSN 2199-2630
issn_series 2199-2622
copyrightSpringer International Publishing Switzerland 2016
The information of publication is updating

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Order vs. Disorder in the Solid State,tinal microbiota, and chronic inflammation. Obesity-related changes in these pathways likely result in alterations in the epigenome of colonic epithelial cells, with the potential to influence cancer development and/or progression.
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Epigenetics, Obesity, and Colon Cancer,tinal microbiota, and chronic inflammation. Obesity-related changes in these pathways likely result in alterations in the epigenome of colonic epithelial cells, with the potential to influence cancer development and/or progression.
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發(fā)表于 2025-3-22 09:10:08 | 只看該作者
Digital Humanities as Theory and Practice,ully characterized, they can be exploited to improve exposure ascertainment. We summarize in this chapter evidence for the influence of environmental exposures on obesity and how epigenetic alterations may contribute to cancers that disproportionately affect minority populations exhibit disparities in incidence and mortality.
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Magdala Gronau,Siegfried Methfesselere is also some suggestion that genes involved in cancer-related pathways and pathways implicated in obesity-related cancers and adipose-mediated inflammation (insulin, IGF-1) demonstrate altered methylation in obese individuals. Thus, obesity appears to influence the formation and progression of BE to EAC via epigenetic mechanisms.
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Book 2016des a valuable resource for students, research investigators and clinicians seeking to better understand these processes as well as a basis for novel translational and transdisciplinary approaches to further elucidate these processes and develop preventive and therapeutic strategies.
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Amniotic Membrane in Ophthalmologyf therapies targeting epigenetic modifiers have been approved by the FDA for cancer treatment, and many others are in clinical trials. Ongoing research is focused on better understanding mechanisms contributing to the altered epigenome, how the altered epigenome contributes to malignant transformati
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