Titlebook: Epidemiology of Alzheimer’s Disease: From Gene to Prevention; Richard Mayeux,Y. Christen Conference proceedings 1999 Springer-Verlag Berli

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,Genetic Factors in Early- and Late-Onset Alzheimer’s Disease, that are involved in the autosomal dominant forms of early-onset AD: the β-amyloid precursor protein gene (APP) and two homologous genes, presenilin 1 (PS-1) and presenilin 2 (PS-2). Mutations in each of these genes have been found to be rare. Screening a population-based sample of patients with ea

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,Heterogeneity in Alzheimer’s Disease: Implications for Epidemiology,s and patterns of clinical symptoms, the genetic underpinnings, neuropathological indices, and responses to pharmacological treatments and interventions. Much of the information published on Alzheimer’s disease has been developed from highly selected patient groups. In the U.S., these patient groups

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LUDE

,Inflammation, Anti-Inflammatory Drugs and Alzheimer’s Disease,in Alzheimer’s disease (AD). Neuritic plaques, a hallmark of AD pathology, have been associated with a host of proteins and acute phase reactants, activated microglia, and complement activation. From these observation, it follows that pharmacological suppression of inflammation may slow the rate of

LUDE

,Socio-Demographic Risk Factors for Dementia and Alzheimer’s Disease in the Paquid Study,rmal and pathological brain aging (Barberger-Gateau et al. 1992). The main objective of Paquid was initially the analysis of the natural history of the cognitive disorders occurring at the very early phase of dementia. However, the baseline data collection and the follow-up of the cohort provided th

BRIDE

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,The Implications of Epidemiological Findings for the Prevention of Alzheimer’s Disease, mainly women. This rise is due to the increase in the prevalence of dementia with age, coupled with the mortality at older ages. As populations age yet further, there will be increases in those in older age groups, who are at even greater risk of dementia. Any serious attempt at prevention of Alzhe

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,The Primary Prevention of Alzheimer’s Disease: Should We Develop Large-Scale Trials?,ng to increase in size, the impact of the disease on public health will continue to grow. Improved treatment for persons who have the disease and support for their families are necessary, but by themselves are insufficient to confront a problem of this magnitude; a strategy for primary prevention of