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Titlebook: Endothelial Signaling in Development and Disease; Mirko HH Schmidt,Stefan Liebner Book 2015 Springer Science+Business Media New York 2015

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發(fā)表于 2025-3-23 10:53:21 | 只看該作者
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發(fā)表于 2025-3-23 15:36:02 | 只看該作者
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發(fā)表于 2025-3-23 21:01:07 | 只看該作者
Schulterendoprothetik beim Trauma, participation of different molecular families that were first identified to have a role in the nervous system. This chapter will therefore focus on the canonical axon guidance cues netrins, slits, semaphorins, and ephrins, and summarize the current knowledge regarding their role in the vascular system during health and disease.
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發(fā)表于 2025-3-24 00:14:34 | 只看該作者
https://doi.org/10.1007/978-3-662-48787-7 inflammation. In this chapter we describe classical vascular functions and discuss novel aspects with regard to immune cell recruitment, as well as recent therapeutic strategies targeting the angiopoietin/Tie system.
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發(fā)表于 2025-3-24 05:21:16 | 只看該作者
Book 2015stics, and pathologies of the vascular system such as angiogenesis, fibroblast growth factor signaling, lymphangiogenesis, junctional signaling, the extracellular matrix, vascular permeability, leukocyte extravasation, axon guidance factors, the angiopoietin system, and chronic obstructive lung dise
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發(fā)表于 2025-3-24 09:00:37 | 只看該作者
Multidirektionale Schulterinstabilit?tdepletion of tetrahydrobiopterin, an essential co-factor for the eNOS enzyme, and deficiency of L-arginine, the eNOS substrate. Reversal of eNOS uncoupling may represent a novel therapeutic strategy for the prevention and treatment of vascular diseases.
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發(fā)表于 2025-3-24 12:54:42 | 只看該作者
Nitric Oxide Synthesis in Vascular Physiology and Pathophysiologydepletion of tetrahydrobiopterin, an essential co-factor for the eNOS enzyme, and deficiency of L-arginine, the eNOS substrate. Reversal of eNOS uncoupling may represent a novel therapeutic strategy for the prevention and treatment of vascular diseases.
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發(fā)表于 2025-3-24 16:48:35 | 只看該作者
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