Titlebook: Endothelial Cell Dysfunctions; Nicolae Simionescu,Maya Simionescu Book 1992 Springer Science+Business Media New York 1992 Pathogene.adapta

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書目名稱Endothelial Cell Dysfunctions影響因子(影響力)




書目名稱Endothelial Cell Dysfunctions影響因子(影響力)學(xué)科排名




書目名稱Endothelial Cell Dysfunctions網(wǎng)絡(luò)公開度




書目名稱Endothelial Cell Dysfunctions網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Endothelial Cell Dysfunctions被引頻次




書目名稱Endothelial Cell Dysfunctions被引頻次學(xué)科排名




書目名稱Endothelial Cell Dysfunctions年度引用




書目名稱Endothelial Cell Dysfunctions年度引用學(xué)科排名




書目名稱Endothelial Cell Dysfunctions讀者反饋




書目名稱Endothelial Cell Dysfunctions讀者反饋學(xué)科排名

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Modulation of Fibronectin and Thrombospondin Synthesis and mRNA Levels by Heparin in Human Endothelicored the potential importance of heparin in the regulation of new vessel formation. . heparin potentiates human umbilical vein endothelial cell (EC)* chemotactic and growth responses to endothelial cell growth factor (ECGF), a member of the fibroblast growth factor (FGF) family of mitogens.. The gr

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Dysfunction of the Release of Endothelium-Derived Relaxing Factories such as hypertension, hyperlipidemia, diabetes, and ischemia (Fig. 1).. Although it has been known for years that endothelial cells undergo morphological changes under these conditions, the functional alterations have only been characterized recently. As the endothelium plays a protective role i

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Endothelial-Leukocyte Adhesion Molecules in Acute Inflammation and Atherogenesisblood; yet, these processes involve different leukocytes, and have distinct locations, chronologies, and consequences. Acute inflammation is a rapid response of the host to local perturbations, such as trauma or bacterial infection, and is typically followed by resolution.. A prominent feature of ac

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