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Titlebook: Endothelial Cell Biology in Health and Disease; Nicolae Simionescu,Maya Simionescu Book 1988 Plenum Press, New York 1988 Vivo.biochemistry

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發(fā)表于 2025-3-21 19:00:42 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Endothelial Cell Biology in Health and Disease
編輯Nicolae Simionescu,Maya Simionescu
視頻videohttp://file.papertrans.cn/310/309935/309935.mp4
圖書封面Titlebook: Endothelial Cell Biology in Health and Disease;  Nicolae Simionescu,Maya Simionescu Book 1988 Plenum Press, New York 1988 Vivo.biochemistry
描述Although blood capillaries were first observed through a flea-lens microscope by Malpighi in 1661,200 more years elapsed before the cellular nature of the vessel wall was conclusively demonstrated. Beginning with the middle of the 19th cen- tury, our knowledge of the histological organization of blood vessels has steadily increased. However, the endothelium, which for a long time was considered to be just an inert barrier lining, had been barely explored until three decades ago. Since then, there has been an upsurge of interest in the fine structure and function of endothelial cells. Intense in vivo and in vitro investigations have revealed that the endothelial cell is a key element in a wide variety of normal activities and diseases. A large number of investigators and laboratories have been attracted to endothelial cell research, thus supporting the expansion of the continuously grow- ing and diversifying field of endotheliology. The number of articles published annually on this subject has increased from a few score at the beginning of the 1970s to more than a thousand in recent years, and an increasing number of journals, books, societies, and symposia focused primarily on the
出版日期Book 1988
關鍵詞Vivo; biochemistry; biology; cell; cell biology; cell growth; developmental biology; electron microscopy; hy
版次1
doihttps://doi.org/10.1007/978-1-4613-0937-6
isbn_softcover978-1-4612-8254-9
isbn_ebook978-1-4613-0937-6
copyrightPlenum Press, New York 1988
The information of publication is updating

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沙發(fā)
發(fā)表于 2025-3-21 22:00:00 | 只看該作者
https://doi.org/10.1007/978-3-322-93836-7ally true since many of the relevant cell surface and metabolic properties of endothelium are inducible, rather than constitutive, and thus may be subject to physiologic regulation and potential pathologic derangement.
板凳
發(fā)表于 2025-3-22 01:41:40 | 只看該作者
地板
發(fā)表于 2025-3-22 08:31:57 | 只看該作者
Ultrastructural Studies of Capillary Endotheliumvities of the individual endothelial cells. And yet there is no comprehensive theory which adequately reconciles the permeability behavior of continuous capillaries with ultrastructural correlates in the capillary wall.
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發(fā)表于 2025-3-22 11:38:41 | 只看該作者
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發(fā)表于 2025-3-22 13:59:44 | 只看該作者
Assembly and Function of the Protein C Anticoagulant Pathway on Endotheliumh the necrotic area is characterized by fibrin deposition in the microvasculature. In several patients examined retrospectively, low levels of protein C have been observed which probably contributed to the coagulopathy..
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發(fā)表于 2025-3-22 18:54:14 | 只看該作者
nature of the vessel wall was conclusively demonstrated. Beginning with the middle of the 19th cen- tury, our knowledge of the histological organization of blood vessels has steadily increased. However, the endothelium, which for a long time was considered to be just an inert barrier lining, had be
8#
發(fā)表于 2025-3-22 23:48:02 | 只看該作者
https://doi.org/10.1007/978-3-642-49864-0 and exhibits a structure and biochemistry subtly differentiated according to the activities of the tissue in which it resides.. As demonstrated by work carried out especially in the last decade, the participation of endothelial cells in the plasma-interstitial fluid exchanges is a more refined and dynamic phenomenon than initially considered.
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發(fā)表于 2025-3-23 01:23:56 | 只看該作者
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發(fā)表于 2025-3-23 09:11:15 | 只看該作者
Endothelium and the Regulation of Coagulation it a central role in procoagulant reactions.. Once the placelet is stimulated, however, there is no turning back: the platelet is an end-stage cell committed to the augmentation of thrombin formation. Thus, the platelet is not a sensitive regulator of anticoagulant and procoagulant events.
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