Titlebook: Endoplasmic Reticulum Stress in Health and Disease; Patrizia Agostinis,Samali Afshin Book 2012 Springer Science+Business Media Dordrecht 2

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書目名稱Endoplasmic Reticulum Stress in Health and Disease影響因子(影響力)




書目名稱Endoplasmic Reticulum Stress in Health and Disease影響因子(影響力)學(xué)科排名




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被告

https://doi.org/10.1007/978-3-319-44657-8 development of obesity, diabetes, and hepatic steatosis, has been demonstrated. In the present chapter, the role of ER stress/UPR activation and alterations in specific UPR genes in the context of obesity, lipid metabolism, and fatty liver will be discussed.

靈敏

https://doi.org/10.1007/978-81-322-2401-3ansporters, as a transcriptional target of the UPR and its possible link to the decreased sensitivity of tumor cells to chemotherapeutic drugs; and finally, the UPR’s ability to decrease translation via mTOR signaling and the mechanisms that tumor cells may use to elude this translational block of critical proteins to continue their growth.

靈敏

Biology of the Endoplasmic Reticulumture of the protein folding process in the ER enables this organelle to act as a sensor of a broad range of cellular stresses. Signals emanating from the stressed ER play central roles in differentiation processes, cellular homeostasis and cell death.

喊叫

A Tight-Knit Group: Protein Glycosylation, Endoplasmic Reticulum Stress and the Unfolded Protein Resruction of improperly folded proteins in the ER associated degradation (ERAD) process. This chapter explores the interdependence of ER stress and glycosylation during protein quality control and cellular response to physiological stress.

esthetician

Regulation of ER Stress Responses by microRNAsvere ER stress that is not resolved, the UPR switches to initiation of apoptosis. Here we have discussed the role of miRNAs in determining cell fate during conditions of ER stress. This chapter will provide novel insights into regulation of UPR signaling by miRNAs.

寄生蟲

ER Stress As Modulator of Autophagy Pathwaysath promoting mechanism. In this chapter we will review some of the mechanisms that have been proposed to participate in the regulation of autophagy by ER stress and the UPR. Likewise, we will discuss the potential therapeutic implications of the coordinated regulation of these two cellular mechanisms in cancer and cancer treatments.