Titlebook: Endometriosis; Pathogenesis and Tre Tasuku Harada Book 2014 Springer Japan 2014 Endometriosis.infertility.oocytes.ovarian cysts.ovaries.pro

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Role of Stem Cells in the Pathogenesis of Endometriosisow indicate that multiple genetic and epigenetic changes (reminiscent of neoplastic processes) are involved in the pathophysiology of endometriosis. Given the molecular similarities between endometriosis and cancer, it is reasonable to apply the “cancer stem cell model” concept to the pathogenesis o

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Macrophages in Pathophysiology of Endometriosis endometriosis is associated with changes in population and functions of various leukocytes, including macrophages. Macrophages are the most abundant cells found in the peritoneal fluid and are the consistent feature of endometriotic lesion. They infiltrate endometriotic lesions where they undergo a

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Roles of Prostaglandin E2 in Endometriosispresence of endometrial-like glands and stroma outside the uterus, commonly on the pelvic peritoneum and ovaries. The major symptoms of endometriosis include pelvic pain, dysmenorrhea, dyspareunia, and infertility. During the past decade, intensive investigations on molecular mechanisms responsible

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Sex Steroids and Endometriosis Estrogen metabolism differs considerably in women with a normal endometrium compared to those with estrogen-dependent uterine diseases, including endometriosis, adenomyosis, or fibromas. Altered expression patterns of estrogen receptor (ER)-α/ER-β, progesterone receptor (PR)-A/PR-B, and 17β-hydroxy

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Aromatase Expression in Endometriosis and Its Significanceon of an endometriotic lesion depends on a sex steroid, estrogen. Aromatase, a key enzyme in estrogen biosynthesis, is highly expressed in the endometriotic tissue, resulting in in situ production of estrogen that, in addition to endocrine estrogen from the ovary, may contribute to the etiology and

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Apoptosis in Endometriosistrium from women with endometriosis has some fundamental differences compared with the normal endometrium of women without endometriosis. The differences could contribute to the survival of regurgitating endometrial cells into the peritoneal cavity and the development of endometriosis. One mechanism